In the published article, there was an error in Table 1 as published. “GADA reactivities to epitopes of GAD65” in Table 1 was improperly named. “N-region” should be corrected to “N-terminal”, and “M-terminal” to “Middle region”. The corrected Table 1 and its caption appear below.
Table 1
| GADA reactivities to epitopes of GAD65 | Young T1DM (n=165) | LADY (n=94) | Old T1DM (n=149) | LADA (n=78) | p value |
|---|---|---|---|---|---|
| C-terminal | 57.6% (95) | 48.9% (46) | 58.4% (87) | 33.3% (26) †††§*** | 0.001 |
| N-terminal | 7.3% (12) | 12.8% (12) | 12.8% (19) | 20.5% (16) †† | 0.031 |
| Middle region | 60.0% (99) | 66.0% (62) | 54.4% (81) | 42.3% (33) †§§ | 0.012 |
| Positive for 1 epitope | 30.9% (51) | 27.7% (26) | 32.9% (49) | 32.1% (25) | 0.855 |
| Positive for at least 2 epitopes | 44.2% (73) | 45.7% (43) | 43.6% (65) | 26.9% (21) †§* | 0.040 |
Prevalence of epitope-specific GAD65Ab in young T1DM, old T1DM, LADY and LADA patients.
Data are expressed as % (n).
GADA, glutamic acid decarboxylase antibody.
When compared with young T1DM patients, †p < 0.05, ††p < 0.01, †††p < 0.001.
When compared with LADY patients, §p < 0.05, §§p < 0.01, §§§p < 0.001.
When compared with old T1DM patients, *p < 0.05, **p < 0.01, ***p < 0.001.
In the published article, there was an error in Figure 1 as published. “Young T1D” should be changed to “young T1DM”, “wereremoved” should be changed to “were removed” and “old T1dM” should changed to “old T1DM”. The corrected Figure 1 and its caption appear below.
Figure 1
In the published article, there was an error. A correction has been made to Materials and Methods, HLA -DR-DQ Genetic Analysis. This sentence previously stated:
“The susceptible HLA haplotypes were DR3 (DRB1*0301-DQA1*0501-DQB1*0201), DR4 (DRB1*0405-DQA1*0303-DQB1*0401), DRB1*0405-DQA1*0301-DQB1*0302, and DR9 (DRB1*0901-DQA1*0303-DQB1*0302) (15, 21–23).”
The corrected sentence appears below:
“The susceptible HLA haplotypes were DR3 (DRB1*0301-DQA1*0501-DQB1*0201), DR4 (DRB1*0405-DQA1*0303-DQB1*0401), DRB1*0405-DQA1*0301-DQB1*0302, and DR9 (DRB1*0901-DQA1*0302-DQB1*0303) (15, 21-23).”
The original article has been updated.
Statements
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
glutamic acid decarboxylase autoantibody, GAD epitopes, HLA, type 1 diabetes (T1D), latent autoimmune diabetes in adults (LADA), latent autoimmune diabetes in youth
Citation
Peng Y, Li X, Xiang Y, Yan X, Zhou H, Tang X, Cheng J, Niu X, Liu J, Ji Q, Ji L, Huang G and Zhou Z (2026) Correction: GAD65 antibody epitopes and genetic background in latent autoimmune diabetes in youth (LADY). Front. Immunol. 17:1966969. doi: 10.3389/fimmu.2026.1966969
Received
13 August 2026
Accepted
18 August 2026
Published
02 September 2026
Volume
17 - 2026
Edited and reviewed by
Hubert M. Tse, University of Alabama at Birmingham, United States
Updates
Copyright
© 2026 Peng, Li, Xiang, Yan, Zhou, Tang, Cheng, Niu, Liu, Ji, Ji, Huang and Zhou.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Gan Huang, huanggan007@csu.edu.cn
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.