About this Research Topic
Oxidative stress and inflammation are the major defense of the cells and body adapt to the environmental conditions. They have been found to be related to the initiation and development of a variety of animal and human diseases, especially in the modern world. There is also an interactive relationship between oxidative stress and inflammation.
Exposure to the environmental risk factors, mitochondrial function and micro-ecological balance would be a break. The reactive oxygen species (ROS) production of mitochondrial is the most important source of cellular ROS in healthy tissues. Mitochondrial dysfunction influences bioenergetics which contributes to the decline of tissue functionality of animal and human beings. Microbiota means the population of microorganisms (bacteria, viruses and fungi) is altered along with oxidative stress and inflammation develop. Growing studies suggest that nutritional strategy possess anti-oxidative and/or anti-inflammatory effects. However, the detailed mechanisms underlying remains largely unknown.
In this Research Topic, we invite investigators to contribute original research articles using in vitro or in vivo models (small or big animals) and studies to the extant body of literature addressing the roles of nutritional strategy in regulation of oxidative stress and inflammation and further explore the cellular and molecular mechanisms underlying their actions.
We welcome various types of manuscripts, including original research and review articles. All manuscripts should be relevant to the following specific themes but not limited to:
• Mitochondria dysfunction in oxidative stress and inflammation
• Microbiota changes in oxidative stress and inflammation
• Crosstalk between oxidative stress and inflammation
• Redox and epigenetic regulation
• Food and feed resources against the risk caused by oxidative stress- and/or inflammation-related disorders
Exposure to the environmental risk factors, mitochondrial function and micro-ecological balance would be a break. The reactive oxygen species (ROS) production of mitochondrial is the most important source of cellular ROS in healthy tissues. Mitochondrial dysfunction influences bioenergetics which contributes to the decline of tissue functionality of animal and human beings. Microbiota means the population of microorganisms (bacteria, viruses and fungi) is altered along with oxidative stress and inflammation develop. Growing studies suggest that nutritional strategy possess anti-oxidative and/or anti-inflammatory effects. However, the detailed mechanisms underlying remains largely unknown.
In this Research Topic, we invite investigators to contribute original research articles using in vitro or in vivo models (small or big animals) and studies to the extant body of literature addressing the roles of nutritional strategy in regulation of oxidative stress and inflammation and further explore the cellular and molecular mechanisms underlying their actions.
We welcome various types of manuscripts, including original research and review articles. All manuscripts should be relevant to the following specific themes but not limited to:
• Mitochondria dysfunction in oxidative stress and inflammation
• Microbiota changes in oxidative stress and inflammation
• Crosstalk between oxidative stress and inflammation
• Redox and epigenetic regulation
• Food and feed resources against the risk caused by oxidative stress- and/or inflammation-related disorders
Keywords: oxidative stress, inflammation, mitochondria, microbiota, nutrition and health
Important Note: All contributions to this Research Topic must be within the scope of the section and journal to which they are submitted, as defined in their mission statements. Frontiers reserves the right to guide an out-of-scope manuscript to a more suitable section or journal at any stage of peer review.
