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Cardiovascular diseases are the leading cause of death globally, accounting for more than 17.9 million death each year or 31 percent of all deaths worldwide. Metabolic cardiomyopathy (MeC) is a special type of cardiovascular disease caused by systematic metabolic disorders inherited or acquired during ...

Cardiovascular diseases are the leading cause of death globally, accounting for more than 17.9 million death each year or 31 percent of all deaths worldwide. Metabolic cardiomyopathy (MeC) is a special type of cardiovascular disease caused by systematic metabolic disorders inherited or acquired during adulthood. It contains a wide spectrum of pathological conditions due to defects in glucose metabolism, fatty acid metabolism, impaired calcium handling, etc. Understanding the molecular mechanism of MeC and developing therapeutic approaches that can improve the outcome of patients with MeC represent the forefront of cardiovascular research.

This research topic will focus on new research in the areas of metabolic damage mechanisms, and intervention strategies. Both basic research and clinical transitional studies will be welcomed. The primary objective is to comprehensively summarize the current perspectives and propose future research directions regarding genetics, diagnosis, staging, classification, mechanisms, new models, new methodologies, etiology or risk factors, prevention, and treatment of MeCs including diabetic cardiomyopathy, myocardial ischemia reperfusion, and hypertension cardiomyopathy, etc.

We encourage researchers to submit Original Research, Reviews, Case Studies, Opinions and Perspectives including, but not limited, to any one or more of the following themes. The following topics illustrate the target subject areas and scope of the project.
1. Molecular Mechanism and/or Pharmacology of Diabetic cardiomyopathy.
2. Molecular Mechanism and/or Pharmacology of myocardial ischemia reperfusion.
3. Molecular Mechanism and/or Pharmacology of hypertension cardiomyopathy.

Keywords: Diabetic cardiomyopathy, myocardial ischemia reperfusion, hypertension, molecular mechanism, pharmacology


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