About this Research Topic
The predominant, continuous, and high-level circulation of a new respiratory pathogen could modify the circulation, evolution, pathogenesis, virulence, and/or epidemiology of other viruses. For example, derived from the 2009 influenza A/H1N1 pandemic, RSV duplicated a segment of its G gene (this generate the ON1 genotype). Another example was the 2020-21 winter season, where there was no influenza virus circulation. We are currently detecting viruses outside of their usual seasons or with changes in their clinical presentations.
The objective of this topic is to evaluate the impact or effect of the predominant circulation of the SARS- CoV-2 virus, during the current pandemic on other viruses. Particularly, we are interested in the effect on genetic evolution, changes in the presentation or severity of diseases, changes in population circulation or changes in epidemiological behaviour.
The approach of the articles for this topic range from genetics of viral population, phylogenetics, phylogeography, phylodynamics, epidemiology evaluation, clinical presentation or severity of diseases and mathematical modelling.
The objective of this topic is to evaluate the impact or effect of the predominant circulation of the SARS- CoV-2 virus, during the current pandemic on other viruses. Particularly, we are interested in the effect on genetic evolution, changes in the presentation or severity of diseases, changes in population circulation or changes in epidemiological behaviour.
The approach of the articles for this topic range from genetics of viral population, phylogenetics, phylogeography, phylodynamics, epidemiology evaluation, clinical presentation or severity of diseases and mathematical modelling.
Keywords: SARS-CoV-2, viral evolution, epidemiology, mathematics, viral interference
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