High-temperature stress (HS) is one of the major environmental threats to agriculture and crop production. It causes an array of morpho-physiological and molecular changes in plants resulting in a significant reduction of growth and yield formation. At the cellular level, HS changes membrane integrity, induces protein denaturation and aggregation, and results in metabolic alterations. Understanding HS signaling and identifying the cellular regulatory mechanisms that promote plant survival and reproduction under high-temperature conditions are therefore crucial for developing climate-resilient crops. The response of plants to HS involves complex protein quality control (PQC) mechanisms, which involve molecular chaperones and autophagy, hormonal alterations, and the formation of transient condensate foci composed of RNA, protein, and metabolites. Although research during the last decades has enhanced our understanding of the plant's response to HS, the knowledge about the molecular-biochemical regulatory processes involved in HS perception, signal transduction, and adaptation remains fragmented.
This Research Topic aims to cover new progress about regulatory mechanisms involved in HS responses in model species and crops. This includes molecular, biochemical and cellular mechanisms related to known and novel players in HS sensing, signaling and adaptation mechanisms. Of particular interest for this topic are reports on the role and regulation of condensates/foci and stress granules forming upon HS and their involvement in stress signaling and adaptation.
This Research Topic aims to provide a framework for addressing relevant queries in relation to HS signaling, signal transduction and tolerance. We invite contributions that fall under, but are not limited to, the following topics:
• Thermosensing
• Novel players in HS signal transduction pathways
• Transcription regulation of HS response
• Role of protein quality control (PQC) mechanisms (e.g., autophagy) in HS response
• Metabolic control of HS response
• Role and composition of condensates in HS signaling
• Role of stress granules in HS signaling
• Adaptation mechanisms to high temperature
Research articles, review articles as well as short communications are invited.
High-temperature stress (HS) is one of the major environmental threats to agriculture and crop production. It causes an array of morpho-physiological and molecular changes in plants resulting in a significant reduction of growth and yield formation. At the cellular level, HS changes membrane integrity, induces protein denaturation and aggregation, and results in metabolic alterations. Understanding HS signaling and identifying the cellular regulatory mechanisms that promote plant survival and reproduction under high-temperature conditions are therefore crucial for developing climate-resilient crops. The response of plants to HS involves complex protein quality control (PQC) mechanisms, which involve molecular chaperones and autophagy, hormonal alterations, and the formation of transient condensate foci composed of RNA, protein, and metabolites. Although research during the last decades has enhanced our understanding of the plant's response to HS, the knowledge about the molecular-biochemical regulatory processes involved in HS perception, signal transduction, and adaptation remains fragmented.
This Research Topic aims to cover new progress about regulatory mechanisms involved in HS responses in model species and crops. This includes molecular, biochemical and cellular mechanisms related to known and novel players in HS sensing, signaling and adaptation mechanisms. Of particular interest for this topic are reports on the role and regulation of condensates/foci and stress granules forming upon HS and their involvement in stress signaling and adaptation.
This Research Topic aims to provide a framework for addressing relevant queries in relation to HS signaling, signal transduction and tolerance. We invite contributions that fall under, but are not limited to, the following topics:
• Thermosensing
• Novel players in HS signal transduction pathways
• Transcription regulation of HS response
• Role of protein quality control (PQC) mechanisms (e.g., autophagy) in HS response
• Metabolic control of HS response
• Role and composition of condensates in HS signaling
• Role of stress granules in HS signaling
• Adaptation mechanisms to high temperature
Research articles, review articles as well as short communications are invited.
