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Optimum endometrial thickness (EMT) before and during gestation are associated with healthy and full-term pregnancies. The menstrual cycle and pregnancy cause changes in EMT and it is understood that the endometrium should not be too thick (endometrial hyperplasia) or too thin in order to facilitate ...

Optimum endometrial thickness (EMT) before and during gestation are associated with healthy and full-term pregnancies. The menstrual cycle and pregnancy cause changes in EMT and it is understood that the endometrium should not be too thick (endometrial hyperplasia) or too thin in order to facilitate successful embryo implant and nourishment. Thus, ultrasound is used to measure the thickness of the endometrium and can impact pregnancy or success of in vitro fertilization (IVF) and intracytoplasmic sperm injection (ICSI).

Factors known to cause endometrial hyperplasia include endometrial cancer; obesity; hormone replacement therapy; high blood pressure; endometrial polyps; diabetes; excessive estrogen production; insufficient progesterone; and/or scar tissue. Causes of a thin endometrium include inflammation; insufficient estrogen; or the structure of the endometrium itself.

The current Research Topic seeks to present Original Research, Reviews, Mini-Reviews or other accepted article types. These may cover, but are not limited to, the following sub-themes:
- Impacts of excessive or insufficient endometrial thickness on conception and pregnancy;
- Factors that unfavorably increase or decrease endometrial thickness;
- Novel uses of endometrial thickness as a clinical monitoring index;
- Endometrial thickness in pregnant women with existing conditions (e.g. PCOS, endometrial cancer, etc.)
- Treatment of high or low endometrial thickness during, or to facilitate, pregnancy.

Keywords: endometrial thickness, pregnancy, endometrium


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