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Obesity is known to be a contributing risk factor for numerous diseases and viral infections, such as diabetes, H1N1 influenza, and respiratory issues, among many others. Throughout the Covid-19 pandemic there has been consistent evidence that obesity is also one of the strongest risk factors and predictors ...

Obesity is known to be a contributing risk factor for numerous diseases and viral infections, such as diabetes, H1N1 influenza, and respiratory issues, among many others. Throughout the Covid-19 pandemic there has been consistent evidence that obesity is also one of the strongest risk factors and predictors for Covid-19 hospitalization as well as severe outcomes and complications. With the rise in obesity around the world, this leaves a significant portion of the world population vulnerable to viral infection, aggravating further the public health crisis.

Numerous mechanisms and hypotheses have recently been suggested to explain the relationship between obesity and the severity of Covid-19. In particular, the contribution of other shared comorbidities such as hypertension, respiratory illness, and cardiovascular disease. Other potential connections may relate to the increased levels of adipose tissue and circulatory cells (E.g., monocytes and leukocytes) and the inflammatory cytokines that they secrete, unbalance in the adipocytokine microenvironment, disturbed immune regulation, and altered metabolic homeostasis in obese patients. Further research is needed to better understand this relationship and provide better treatment options.

The goal of this Research Topic is to compile novel research and reviews discussing the relationship between obesity and Covid-19. We welcome original research, reviews, mini reviews, perspectives, and systematic review articles.

*For articles relating to other aspects of Covid-19 and endocrine research, please see our related Research Topics
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Keywords: covid-19, inflammation, hypertension, adipocytokine, metabolic homeostasis, adipose tissue


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