The production and perception of speech is the most effective way for human communication. This ability is supported by complex neural mechanisms that provide the sensorimotor interface to monitor auditory feedback for fluent production and comprehension of speech. The critical involvement of the auditory system in speech is supported by evidence from altered feedback studies demonstrating compensatory speech behavior in response to external manipulations of online auditory input. In addition, other studies have revealed the effects of auditory disorders on degraded ability for speech production and perception. However, despite advances on characterizing the behavioral influences of auditory disorders on speech, our knowledge about the neurobiological foundations of such effects has remained elusive.
The present topic aims at inviting research studies to advance the understanding of auditory disorders and their influence on the neural mechanisms of speech production and perception. The primary goal is to highlight neuroscience research that tackle challenging questions on how peripheral and central neural deficits in the auditory system would affect sensorimotor mechanisms implicated in speech production and perception. The broad-scope goal of the research topic is to promote knowledge for advancing the theoretical foundations of speech production as a model system as well as developing new technologies for the diagnosis and treatment of neurological conditions leading to speech sensorimotor disorders.
The research topic invites studies from a wide range of theoretical, empirical, and clinical research aiming at understanding the effects of auditory neural disorders on speech production and perception mechanisms. Preferred topics include but are not limited to areas listed below:
• Auditory processing disorder
• Hearing loss
• Auditory neuropathy
• Cochlear implant
• Auditory hallucination
• Autism spectrum disorder
• Alzheimer’s disease
• Parkinson’s disease
• Huntington’s disease
• Stuttering
• Tinnitus
• Stroke
The production and perception of speech is the most effective way for human communication. This ability is supported by complex neural mechanisms that provide the sensorimotor interface to monitor auditory feedback for fluent production and comprehension of speech. The critical involvement of the auditory system in speech is supported by evidence from altered feedback studies demonstrating compensatory speech behavior in response to external manipulations of online auditory input. In addition, other studies have revealed the effects of auditory disorders on degraded ability for speech production and perception. However, despite advances on characterizing the behavioral influences of auditory disorders on speech, our knowledge about the neurobiological foundations of such effects has remained elusive.
The present topic aims at inviting research studies to advance the understanding of auditory disorders and their influence on the neural mechanisms of speech production and perception. The primary goal is to highlight neuroscience research that tackle challenging questions on how peripheral and central neural deficits in the auditory system would affect sensorimotor mechanisms implicated in speech production and perception. The broad-scope goal of the research topic is to promote knowledge for advancing the theoretical foundations of speech production as a model system as well as developing new technologies for the diagnosis and treatment of neurological conditions leading to speech sensorimotor disorders.
The research topic invites studies from a wide range of theoretical, empirical, and clinical research aiming at understanding the effects of auditory neural disorders on speech production and perception mechanisms. Preferred topics include but are not limited to areas listed below:
• Auditory processing disorder
• Hearing loss
• Auditory neuropathy
• Cochlear implant
• Auditory hallucination
• Autism spectrum disorder
• Alzheimer’s disease
• Parkinson’s disease
• Huntington’s disease
• Stuttering
• Tinnitus
• Stroke