Endocrine function may be adversely affected by pesticides and environmental pollutants. The idea that these chemicals act as endocrine disruptors, was initially introduced by Rachel Louise Carson – a scientist and ecologist – in the early 1960s in her work 'Silent Spring'. To date countless publications have discussed this subject from different points of view. Environmental pollution and global climate change, have consensual action on endocrine functions and metabolism, and they may account for the increasing global incidence of metabolic diseases and reproductive disorders.
The so-called One Health concept is an approach to optimize health outcomes by analysing close connections among people, animals and environment, with emphasis on environmental hazards as early indicators. It is notable, that companion animals suffer from analogous diseases to human population. It is plausible, that obesity status, metabolic fitness, the incidence of endocrine diseases and impaired reproduction of among companion animals of a given human population might be powerful early indicators of the respective diseases in humans. Disorders induced by environmental pollution include a wide spectrum of metabolic disorders, such as obesity, endocrinopathies, various malignancies – however, the different resistance and sensitivity of different companion animals and humans are yet to be defined.
Numerous publications discuss the short- and long-term effect of environmental pollution, food preservatives, household chemicals on cell functions and metabolism. However, comparative aspects of endocrine disruptors are not well defined and potential species-specific indicators are unexplored. This research topic aims to discover the analogue endocrine processes in different subjects and similarities in morbidity and incidence of identical disorders.
Manuscripts from field or laboratory research, statistical analysis of population surveys, and demonstration of comparative aspects are welcome. Subjects that could be covered are, but not limited to:
• effects of environmental pollution on metabolism
• endocrinopathies in connection to food preservatives
• appearance of endocrine disruptors at different points of metabolism
• incidence of endocrinopathies and/or metabolic syndromes at population level
• alteration of reproduction impaired by environmental pollution
Endocrine function may be adversely affected by pesticides and environmental pollutants. The idea that these chemicals act as endocrine disruptors, was initially introduced by Rachel Louise Carson – a scientist and ecologist – in the early 1960s in her work 'Silent Spring'. To date countless publications have discussed this subject from different points of view. Environmental pollution and global climate change, have consensual action on endocrine functions and metabolism, and they may account for the increasing global incidence of metabolic diseases and reproductive disorders.
The so-called One Health concept is an approach to optimize health outcomes by analysing close connections among people, animals and environment, with emphasis on environmental hazards as early indicators. It is notable, that companion animals suffer from analogous diseases to human population. It is plausible, that obesity status, metabolic fitness, the incidence of endocrine diseases and impaired reproduction of among companion animals of a given human population might be powerful early indicators of the respective diseases in humans. Disorders induced by environmental pollution include a wide spectrum of metabolic disorders, such as obesity, endocrinopathies, various malignancies – however, the different resistance and sensitivity of different companion animals and humans are yet to be defined.
Numerous publications discuss the short- and long-term effect of environmental pollution, food preservatives, household chemicals on cell functions and metabolism. However, comparative aspects of endocrine disruptors are not well defined and potential species-specific indicators are unexplored. This research topic aims to discover the analogue endocrine processes in different subjects and similarities in morbidity and incidence of identical disorders.
Manuscripts from field or laboratory research, statistical analysis of population surveys, and demonstration of comparative aspects are welcome. Subjects that could be covered are, but not limited to:
• effects of environmental pollution on metabolism
• endocrinopathies in connection to food preservatives
• appearance of endocrine disruptors at different points of metabolism
• incidence of endocrinopathies and/or metabolic syndromes at population level
• alteration of reproduction impaired by environmental pollution
