In the original article, there were some mistake in the Legends for FIGURE 2 | Meta-analysis with a fixed effects model for the association between the 3′-UTR VNTR in SLC6A3 and COPD susceptibility and FIGURE 3 | Meta-analysis with a fixed effects model for the association between the 3′-UTR VNTR in SLC6A3 and COPD susceptibility in Asian and Western populations as published. The “COPD” in the legends of Figures 2 and 3 should be “PD.” The correct legend appears below.
FIGURE 2 | Meta-analysis with a fixed effects model for the association between the 3′-UTR VNTR in SLC6A3 and PD susceptibility.
FIGURE 3 | Meta-analysis with a fixed effects model for the association between the 3′-UTR VNTR in SLC6A3 and PD susceptibility in Asian and Western populations.
Additionally, there were some minor formatting errors in References. Following references: Chang et al., 2018; Wang et al., 2000; Zhang et al., 2000; Zhao et al., 2004 as “(chinese),” should be “in Chinese” And the for reference: Lin et al., 2003, “Lin, J.-J., Yueh, K.-C., Chang, D.-C., Chang, C.-Y., Yeh, Y.-H., and Lin, S.-Z. (2003). The Homozygote 10-copy Genotype of Variable Number Tandem Repeat Dopamine Transporter Gene May Confer protection against Parkinson’s Disease “for Male, but “Not to Female Patients. J. Neurol. Sci. 209, 87–92. doi: 10.1016/s0022-510x(03)00002-9, it should be Lin, J. J., Yueh, K. C., Chang, D. C., Chang, C. Y., Yeh, Y. H., and Lin, S. Z. (2003). The homozygote 10-copy genotype of variable number tandem repeat dopamine transporter gene may confer protection against Parkinson’s disease for male, but not to female patients. J. Neurol. Sci. 209, 87–92. doi: 10.1016/s0022-510x(03)00002-9.”
Finally, Figure 5 was incorrectly cited in the Discussion section. A correction has been made to Section: Discussion, Paragraph 1:
“This meta-analysis assessed the association between the 10R allele of the 3′-UTR VNTR in the SLC6A3 gene and PD, and it included a total of 18 published studies. In general, our findings suggested that the 10R alleles and 10R/10R and 10R/10R + 10R/9R genotypes of the VNTR polymorphism in theSLC6A3 gene confer protection against PD. The 10R alleles and 10R/10R genotype results were replicated in Asian populations, and the 10R/9R genotype was associated with an increased risk of PD in Asian populations. The current meta-analysis confirmed most of the previous findings showing that the 10R allele of the 3′-UTR VNTR in the SLC6A3 gene may be a protective factor in susceptibility to PD.”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Statements
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
Parkinson’s disease, Slc6a3, dopamine transporter, variable number of tandem repeats, metaanalysis
Citation
Zeng Q, Ning F, Gu S, Zeng Q, Chen R, Peng L, Zou D, Ma G and Wang Y (2021) Corrigendum: The 10-Repeat 3′-UTR VNTR Polymorphism in the SLC6A3 Gene May Confer Protection Against Parkinson’s Disease: A Meta-Analysis. Front. Genet. 12:789112. doi: 10.3389/fgene.2021.789112
Received
04 October 2021
Accepted
04 October 2021
Published
21 October 2021
Approved by
Frontiers Editorial Office, Frontiers Media SA, Switzerland
Volume
12 - 2021
Updates
Copyright
© 2021 Zeng, Ning, Gu, Zeng, Chen, Peng, Zou, Ma and Wang.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Dehua Zou, 757384895@qq.com; Guoda Ma, sihan1107@126.com; Yajun Wang, wangyajuny1977@aliyun.com
†These authors have contributed equally to this work
This article was submitted to Statistical Genetics and Methodology, a section of the journal Frontiers in Genetics
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.