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CORRECTION article

Front. Aging Neurosci., 05 August 2025

Sec. Cellular and Molecular Mechanisms of Brain-aging

Volume 17 - 2025 | https://doi.org/10.3389/fnagi.2025.1670545

Correction: Optineurin overexpression ameliorates neurodegeneration through regulating neuroinflammation and mitochondrial quality in a murine model of amyotrophic lateral sclerosis

This article is a correction to:

  1. Optineurin overexpression ameliorates neurodegeneration through regulating neuroinflammation and mitochondrial quality in a murine model of amyotrophic lateral sclerosis

    1. Read original article
Shumin Zhao,Shumin Zhao1,2Ranran Chen,Ranran Chen1,2Yi AnYi An1Yali Zhang,Yali Zhang1,2Cheng MaCheng Ma3Ying Gao,Ying Gao1,2Yanchao Lu,Yanchao Lu1,2Fei Yang,Fei Yang2,4Xue Bai, &#x;Xue Bai1,2*†Jingjing Zhang,, &#x;Jingjing Zhang1,2,3*†
  • 1Department of Neurology, Chifeng Municipal Hospital, Chifeng, China
  • 2Chifeng Clinical Medical College of Inner Mongolia Medical University, Chifeng, China
  • 3Medical Research Center, Chifeng Municipal Hospital, Chifeng, China
  • 4Intensive Care Unit, Chifeng Municipal Hospital, Chifeng, China

A Correction on
Optineurin overexpression ameliorates neurodegeneration through regulating neuroinflammation and mitochondrial quality in a murine model of amyotrophic lateral sclerosis

by Zhao, S., Chen, R., An, Y., Zhang, Y., Ma, C., Gao, Y., Lu, Y., Yang, F., Bai, X., and Zhang, J. (2025). Front. Aging Neurosci. 17:1522073. doi: 10.3389/fnagi.2025.1522073

In the published article, there was an error in the Funding statement. The grant number for the funding source “Natural Science Foundation of Inner Mongolia Autonomous Region” was incorrectly listed as the application code (2022SHZR1527) when it should have instead been (2022MS08048). The corrected Funding statement and code appears below.

Funding

The author(s) declare that financial support was received for the research and/or publication of this article. This study was supported by the National Natural Science Foundation of China (82160320), the Central Government Guides Local Science and Technology Development Fund Projects (2021ZY0010), Natural Science Foundation of Inner Mongolia Autonomous Region (2022MS08048), and the Inner Mongolia Medical University Science Technology Million Project Joint Project [YKD2018KJBW(LH)095].

The original article has been updated.

Publisher's note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: amyotrophic lateral sclerosis, apoptosis, mitochondrial quality, neuroinflammation, optineurin

Citation: Zhao S, Chen R, An Y, Zhang Y, Ma C, Gao Y, Lu Y, Yang F, Bai X and Zhang J (2025) Correction: Optineurin overexpression ameliorates neurodegeneration through regulating neuroinflammation and mitochondrial quality in a murine model of amyotrophic lateral sclerosis. Front. Aging Neurosci. 17:1670545. doi: 10.3389/fnagi.2025.1670545

Received: 21 July 2025; Accepted: 23 July 2025;
Published: 05 August 2025.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2025 Zhao, Chen, An, Zhang, Ma, Gao, Lu, Yang, Bai and Zhang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Xue Bai, MTAzNjUxNjY2OEBxcS5jb20=; Jingjing Zhang, NDU1OTA4NzdAcXEuY29t

†These authors have contributed equally to this work

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.