GENERAL COMMENTARY article

Front. Cell. Neurosci., 19 May 2014

Sec. Cellular Neurophysiology

Volume 8 - 2014 | https://doi.org/10.3389/fncel.2014.00130

Corrigendum: Activation of transient receptor potential vanilloid 4 increases NMDA-activated current in hippocampal pyramidal neurons

  • 1. Department of Physiology, Nanjing Medical University Nanjing, China

  • 2. State Key Laboratory of Reproductive Medicine, Department of Histology and Embryology, Nanjing Medical University Nanjing, China

The dosage of HC-067047 used in the article by Li et al. () should be “10 μ M/2 μ l/mouse.” In addition, in appendix, the perfusion solution for recording EPSCs should be composed of (in mM): NaCl 126, CaCl2 1, KCl 2.5, MgCl2 1, NaHCO3 26, KH2PO4 1.25, D-glucose 20 and bicuculline 0.01.

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Conflict of interest

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

References

Summary

Keywords

TRPV4, NMDA receptor, NR2B subunit, phosphorylation, excitotoxity, cerebral ischemia

Citation

Li L, Qu W, Zhou L, Lu Z, Jie P, Chen L and Chen L (2014) Corrigendum: Activation of transient receptor potential vanilloid 4 increases NMDA-activated current in hippocampal pyramidal neurons. Front. Cell. Neurosci. 8:130. doi: 10.3389/fncel.2014.00130

Received

25 April 2014

Accepted

26 April 2014

Published

19 May 2014

Volume

8 - 2014

Edited and reviewed by

Dieter Wicher, Max Planck Institute for Chemical Ecology, Germany

Copyright

*Correspondence:

This article was submitted to the journal Frontiers in Cellular Neuroscience.

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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