In the original article, there was a mistake in the legend for Figure 1 as published. We neglected to include the citation for the figure from which our figure was adapted and modified. The correct figure legend appears below.
Figure 1. Microglia, like Janus, the two-faced Roman god of beginnings and transitions, display two sides—physiological as well as pathological. While microglial cell activation participates in surveillance that functions to maintain homeostasis and promote synaptic maturation, prolonged exposure to pathogen activators or in settings of systemic inflammation, as may occur in conditions such as diabetes or obesity, can culminate in a state of chronic, non-resolving neuroinflammation. Ultimately, these responses will provoke functional and structural changes and neuronal cell death (neurodegeneration). [Adapted and modified from Heneka et al. (2015). Neuroinflammation in Alzheimer's disease (Figure 1)].
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
References
1
HenekaM. T.CarsonM. J.El KhouryJ.LandrethG. E.BrosseronF.FeinsteinD. L.et al. (2015). Neuroinflammation in Alzheimer's disease. Lancet Neurol.14, 388–405. 10.1016/S1474-4422(15)70016-5
Summary
Keywords
inflammation, mast cells, microglia, astrocytes, oligodendrocytes, neuro-immune, crosstalk, palmitoylethanolamide
Citation
Skaper SD, Facci L, Zusso M and Giusti P (2020) Corrigendum: An Inflammation-Centric View of Neurological Disease: Beyond the Neuron. Front. Cell. Neurosci. 13:578. doi: 10.3389/fncel.2019.00578
Received
12 December 2019
Accepted
16 December 2019
Published
03 February 2020
Approved by
Frontiers Editorial Office, Frontiers Media SA, Switzerland
Volume
13 - 2019
Updates
Copyright
© 2020 Skaper, Facci, Zusso and Giusti.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Morena Zusso morena.zusso@unipd.it
This article was submitted to Cellular Neuropathology, a section of the journal Frontiers in Cellular Neuroscience
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