In the published article, there was an error in the Funding statement. The “Euro Bioimaging Project Roadmap/ESFRI from European Commission” funding was accidentally omitted. The corrected Funding statement appears below.
Funding
This study was funded by Gruppo Famiglie Dravet Onlus, Progetto di Ricerca 2017 to GL, European Union's Horizon 2020 research and innovation program EU-Glia PhD under the Marie Sklodowska-Curie grant agreement No. 722053 to GC, and Euro Bioimaging Project Roadmap/ESFRI from European Commission.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Publisher's note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
extrasynaptic GABA receptor A, delta subunits, GFAP - glial fibrillary acidic protein, Iba-1, SCN1A encephalopathy, Nav1.1 channel
Citation
Goisis RC, Chiavegato A, Gomez-Gonzalo M, Marcon I, Requie LM, Scholze P, Carmignoto G and Losi G (2022) Corrigendum: GABA tonic currents and glial cells are altered during epileptogenesis in a mouse model of Dravet syndrome. Front. Cell. Neurosci. 16:1066985. doi: 10.3389/fncel.2022.1066985
Received
11 October 2022
Accepted
12 October 2022
Published
20 October 2022
Approved by
Frontiers Editorial Office, Frontiers Media SA, Switzerland
Volume
16 - 2022
Updates
Copyright
© 2022 Goisis, Chiavegato, Gomez-Gonzalo, Marcon, Requie, Scholze, Carmignoto and Losi.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Gabriele Losi gabriele.losi@cnr.it
†Present address: Gabriele Losi, Institute of Nanoscience, National Research Council (CNR-NANO), Modena, Italy
This article was submitted to Cellular Neuropathology, a section of the journal Frontiers in Cellular Neuroscience
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.