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CORRECTION article

Front. Genet.

Sec. Cancer Genetics and Oncogenomics

Volume 16 - 2025 | doi: 10.3389/fgene.2025.1684479

Correction: FEZF1-AS1 drives autophagy-mediated progression of colon cancer and reduced chemosensitivity through inhabiting PI3K/AKT/mTOR signaling pathway

Provisionally accepted
Xiaoping YangXiaoping Yang1Zuohui YuanZuohui Yuan2Lingzhu GouLingzhu Gou1Long ChengLong Cheng1Zirui WangZirui Wang3Pingfan WuPingfan Wu4Wang XiaochunWang Xiaochun2Xueni MaXueni Ma1Tiantian MaTiantian Ma5Yi YuYi Yu1Zhiping WuZhiping Wu1Dekui ZhangDekui Zhang1*
  • 1Lanzhou University Second Hospital, Lanzhou, China
  • 2Gansu Provincial Hospital, Lanzhou, China
  • 3The No 2 People's Hospital of Lanzhou, Lanzhou, China
  • 4South China University of Technology, Guangzhou, China
  • 5Xi An Chest Hospital, Xi An, China

This article is a correction to:

  1. FEZF1-AS1 drives autophagy-mediated progression of colon cancer and reduced chemosensitivity through inhabiting PI3K/AKT/mTOR signaling pathway

    1. Read original article

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Keywords: FEZF1-AS1, Colon Cancer, Autophagy, PI3K/AKT/mTOR, chemosensitivity

Received: 12 Aug 2025; Accepted: 05 Sep 2025.

Copyright: © 2025 Yang, Yuan, Gou, Cheng, Wang, Wu, Xiaochun, Ma, Ma, Yu, Wu and Zhang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

* Correspondence: Dekui Zhang, Lanzhou University Second Hospital, Lanzhou, China

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

Supplementary Material