CORRECTION article

Front. Mol. Neurosci., 23 February 2023

Sec. Neuroplasticity and Development

Volume 16 - 2023 | https://doi.org/10.3389/fnmol.2023.1162275

Corrigendum: Neural mechanism underlies CYLD modulation of morphology and synaptic function of medium spiny neurons in dorsolateral striatum

  • 1. School of Life Sciences, South China Normal University, Guangzhou, China

  • 2. School of Life Sciences, Guangzhou University, Guangzhou, China

  • 3. South China Normal University-Panyu Central Hospital Joint Laboratory of Translational Medical Research, Panyu Central Hospital, Guangzhou, China

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In the published article, there was an error in the legend for “Figure 1” as published. “(H) Bar graphs showing more stubby spines and varicosity, but fewer mushroom spines and filopodia, in Cyld−/− MSNs than in Cyld+/+ MSNs.” The corrected legend appears below.

(H) Bar graphs showing more stubby spines and varicosity, but fewer mushroom spines, in Cyld−/− MSNs than in Cyld+/+ MSNs.”

In the published article, there was an error. “P-value error for the main effect of distance in Figure 1E.”

A correction has been made to section 3. Results, “3.1. CYLD affects the morphology and physiological features of MSNs,” [Paragraph 1]. This sentence previously stated:

“main effect of distance, F(3.202,153.3) = 202.200, p = 0.188”

The corrected sentence appears below:

“main effect of distance, F(3.202,153.3) = 202.200, p < 0.0001”

In the published article, there was an error. “Misspelling on a word “GluA2”.”

A correction has been made to section 4. Discussion, “The last paragraph.” This sentence previously stated:

“CYLD deficiency causes an increase in K63-linked ubiquitination of GluA1 and GluA2, resulting in reduced GluA1 and GluA1 surface levels and therefore reduced AMPAR-dependent synaptic transmission in MSNs, which is associated with altered DHPG-and HFS-LTD.”

The corrected sentence appears below:

“CYLD deficiency causes an increase in K63-linked ubiquitination of GluA1 and GluA2, resulting in reduced GluA1 and GluA2 surface levels and therefore reduced AMPAR-dependent synaptic transmission in MSNs, which is associated with altered DHPG-and HFS-LTD.”

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

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Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

CYLD, AMPAR, GluA1, GluA2, K63-linked ubiquitination, synaptic transmission, long-term depression, dorsolateral striatum

Citation

Tan S-y, Jiang J-x, Huang H-x, Mo X-p, Feng J-r, Chen Y, Yang L and Long C (2023) Corrigendum: Neural mechanism underlies CYLD modulation of morphology and synaptic function of medium spiny neurons in dorsolateral striatum. Front. Mol. Neurosci. 16:1162275. doi: 10.3389/fnmol.2023.1162275

Received

09 February 2023

Accepted

13 February 2023

Published

23 February 2023

Volume

16 - 2023

Edited and reviewed by

Victor Anggono, The University of Queensland, Australia

Updates

Copyright

*Correspondence: Li Yang ✉ Cheng Long ✉

This article was submitted to Neuroplasticity and Development, a section of the journal Frontiers in Molecular Neuroscience

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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