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CORRECTION article

Front. Mol. Neurosci., 29 September 2025

Sec. Pain Mechanisms and Modulators

Volume 18 - 2025 | https://doi.org/10.3389/fnmol.2025.1707410

Correction: Endoglin alleviates neuropathic pain by protecting the blood–spinal cord barrier through the TGF-β/Smad2 signaling pathway


SiJing Liao,,&#x;SiJing Liao1,2,3Fei-Ran Zhou&#x;Fei-Ran Zhou3Qun LiQun Li3Tao JinTao Jin4Gui-Fang XiangGui-Fang Xiang5Xin WangXin Wang3Yan LiuYan Liu3Hq ZhuHq Zhu3Qing Liu,
Qing Liu3,5*Yuexin Liu
Yuexin Liu6*Ying Zhang,
Ying Zhang3,7*
  • 1Traditional Chinese Medicine Hospital of Meishan, Meishan, China
  • 2The Affiliated Hospital of Southwest Medical University, Luzhou, China
  • 3The Affiliated Traditional Chinese Medicine Hospital, Southwest Medical University, Luzhou, China
  • 4Suining First People's Hospital, Suining, China
  • 5Hejiang Traditional Chinese Medicine Hospital, Luzhou, China
  • 6Zhongshan Hospital, Fudan University, Shanghai, China
  • 7Luzhou Key Laboratory of Research for Integrative on Pain and Perioperative Organ Protection, Luzhou, China

A Correction on
Endoglin alleviates neuropathic pain by protecting the blood–spinal cord barrier through the TGF-β/Smad2 signaling pathway

by by Liao, S., Zhou, F. -R., Li, Q., Jin, T., Xiang, G. -F., Wang, X., Liu, Y., Zhu, H., Liu, Q., Liu, Y., and Zhang, Y. (2025). Front. Mol. Neurosci. 18:1603619. doi: 10.3389/fnmol.2025.1603619

Affiliation 1. Traditional Chinese Medicine Hospital of Meishan, Meishan, China was erroneously given as 1. Traditional Chinese Medicine Hospital of Meishan, Meisham, China.

Affiliation 3. The Affiliated Traditional Chinese Medicine Hospital, Southwest Medical University, Luzhou, China was erroneously given as 3. Affiliated Traditional Chinese Medicine Hospital, Southwest Medical University, Luzhou, China.

Affiliation 5. Hejiang Traditional Chinese Medicine Hospital, Luzhou, China was erroneously given as 5. Hejiang County Traditional Chinese Medicine Hospital, Luzhou, China.

The original version of this article has been updated.

Publisher's note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: endoglin, TGF-β/Smad2, neuropathic pain, blood–spinal cord barrier, endothelial cell

Citation: Liao S, Zhou F-R, Li Q, Jin T, Xiang G-F, Wang X, Liu Y, Zhu H, Liu Q, Liu Y and Zhang Y (2025) Correction: Endoglin alleviates neuropathic pain by protecting the blood–spinal cord barrier through the TGF-β/Smad2 signaling pathway. Front. Mol. Neurosci. 18:1707410. doi: 10.3389/fnmol.2025.1707410

Received: 17 September 2025; Accepted: 18 September 2025;
Published: 29 September 2025.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2025 Liao, Zhou, Li, Jin, Xiang, Wang, Liu, Zhu, Liu, Liu and Zhang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Qing Liu, MTEwNTg1OTM2OEBxcS5jb20=; Yuexin Liu, bGl1Lnl1ZXhpbkB6cy1ob3NwaXRhbC5zaC5jbg==; Ying Zhang, emhhbmd5aW5nMDIxMjEwQDE2My5jb20=

These authors have contributed equally to this work and share first authorship

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.