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CORRECTION article

Front. Ophthalmol., 01 December 2023

Sec. Glaucoma

Volume 3 - 2023 | https://doi.org/10.3389/fopht.2023.1332312

Corrigendum: Glaucoma and microglia-induced neuroinflammation

  • 1. Department of Ophthalmology, Tohoku University Graduate School of Medicine, Sendai, Japan

  • 2. Department of Ophthalmic Imaging and Information Analytics, Tohoku University Graduate School of Medicine, Sendai, Japan

  • 3. Taylor Family Institute for Innovative Psychiatric Research, Washington University School of Medicine, St. Louis, MO, United States

  • 4. Center for Brain Research in Mood Disorders, Washington University School of Medicine, St. Louis, MO, United States

  • 5. Department of Psychiatry, Washington University School of Medicine, St. Louis, MO, United States

  • 6. Department of Advanced Ophthalmic Medicine, Tohoku University Graduate School of Medicine, Sendai, Japan

  • 7. Department of Retinal Disease Control, Tohoku University Graduate School of Medicine, Sendai, Japan

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Incorrect Reference

In the published article, one of the references has been retracted. This article has been removed, and replaced with the following article: Tremblay MÈ, Stevens B, Sierra A, Wake H, Bessis A, Nimmerjahn A. The role of microglia in the healthy brain. J Neurosci (2011) 31(45):16064-9. doi: 10.1523/JNEUROSCI.4158-11.2011

The article text has also been updated to reflect this change. The first paragraph of Section 2, Functions of Microglia in the Retina, previously read as follows:

“Microglia are thought to derive from monocytes that enter the retina from the blood stream during development, and dynamically move their cellular projections even under physiological conditions (12), making physical contact with neurons and synapses and performing synaptic pruning to remove unnecessary synapses. In the development of excitatory circuits, synaptic pruning, by which extrasynaptic connections are eliminated by microglia, is thought to occur when complement C1q is expressed at synapses by TGF-β secreted from astrocytes (13).”

The corrected sentence appears below:

“Microglia are thought to derive from monocytes that enter the retina from the blood stream during development, and dynamically move their cellular projections even under physiological conditions (12), making physical contact with neurons and synapses and performing synaptic pruning to remove unnecessary synapses (13).”

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Statements

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

glaucoma, neuroinflammation, microglia, NOD-like receptor pyrin domain containing 3 inflammasome, retinal ganglion cell damage

Citation

Ishikawa M, Izumi Y, Sato K, Sato T, Zorumski CF, Kunikata H and Nakazawa T (2023) Corrigendum: Glaucoma and microglia-induced neuroinflammation. Front. Ophthalmol. 3:1332312. doi: 10.3389/fopht.2023.1332312

Received

02 November 2023

Accepted

09 November 2023

Published

01 December 2023

Volume

3 - 2023

Edited and reviewed by

Youichi Shinozaki, Tokyo Metropolitan Institute of Medical Science, Japan

Updates

Copyright

*Correspondence: Makoto Ishikawa,

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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