RETRACTION article

Front. Physiol., 04 August 2022

Sec. Integrative Physiology

Volume 13 - 2022 | https://doi.org/10.3389/fphys.2022.1006583

Retraction: Mitofusin-2 enhances mitochondrial contact with the endoplasmic reticulum and promotes diabetic cardiomyopathy

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The Publisher retracts the cited article.

Following publication, the publisher uncovered evidence that false identities were used in the peer-review process. The assignment of fake reviewers was confirmed by an investigation, conducted in accordance with Frontiers’ policies and the Committee on Publication Ethics (COPE) guidelines.

The investigation also uncovered concerns about the presentation and validity of the data in the article that normally would have led to a rejection. When contacted, the authors failed to provide a data set that adequately supports the reported conclusions.

The authors have not responded to correspondence regarding this retraction.

This retraction was approved by the Chief Editors of Frontiers in Physiology and the Chief Executive Editor of Frontiers.

Summary

Keywords

MFN2, mitochondria-ER contact, Mitochondria, ER, Apoptosis

Citation

Frontiers Editorial Office (2022) Retraction: Mitofusin-2 enhances mitochondrial contact with the endoplasmic reticulum and promotes diabetic cardiomyopathy. Front. Physiol. 13:1006583. doi: 10.3389/fphys.2022.1006583

Received

29 July 2022

Accepted

02 August 2022

Published

04 August 2022

Approved by

Geoffrey A. Head, Baker Heart and Diabetes Institute, Australia

Volume

13 - 2022

Updates

Copyright

*Correspondence: Frontiers Editorial Office,

This article was submitted to Integrative Physiology, a section of the journal Frontiers in Physiology

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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