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ORIGINAL RESEARCH article

Front. Aging Neurosci.

Sec. Alzheimer's Disease and Related Dementias

Associations of anticholinergic burden of medication with cognitive decline and longitudinal brain atrophy in the Alzheimer's disease spectrum

  • 1. German Center of Neurodegenerative Diseases (DZNE), Rostock/Greifswald, Rostock, Germany

  • 2. Department of Psychosomatic Medicine, University Medicine Rostock, Rostock, Germany

  • 3. German Center of Neurodegenerative Diseases (DZNE) Rostock/Greifswald, Rostock, Germany

  • 4. Charite - Universitatsmedizin Berlin Klinik fur Psychiatrie und Psychotherapie Campus Charite Mitte, Berlin, Germany

  • 5. Department of Psychiatry and Psychotherapy, Charité, Berlin, Germany

  • 6. German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany

  • 7. German Center for Neurodegenerative Diseases (DZNE), Goettingen, Germany

  • 8. Department of Psychiatry and Psychotherapy, University Medical Center Goettingen, University of Goettingen, Goettingen, Germany

  • 9. German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany

  • 10. Department of Psychiatry, University of Cologne, Medical Faculty, Cologne, Germany

  • 11. German Center for Neurodegenerative Diseases (DZNE), Magdeburg, Germany

  • 12. Institute of Cognitive Neurology and Dementia Research (IKND), Otto-von-Guericke University, Magdeburg, Germany

  • 13. Department of Radiology, Ludwig Maximilian University Hospital, Munich, Germany

  • 14. German Center for Neurodegenerative Diseases (DZNE), Munich, Germany

  • 15. Department of Psychiatry and Psychotherapy, University Hospital, LMU Munich, Munich, Germany

  • 16. German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany

  • 17. Department of Psychiatry and Psychotherapy, University of Tübingen, Tübingen, Germany

  • 18. Research Division, Federal Institute for Drugs and Medical Devices, Bonn, Germany

  • 19. Center for Translational Medicine, Medical Faculty, University of Bonn, Bonn, Germany

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Abstract

Abstract Background: Anticholinergic side effects of pharmacological treatment are a risk factor for cognitive decline in older people. Here, we aimed to assess the effect of anticholinergic burden of treatment on longitudinal rates of cognitive change and atrophy in functionally related brain regions in people from the Alzheimer's disease (AD) spectrum. Methods: We determined associations of anticholinergic burden of pharmacological treatment with rates of global cognition, episodic memory and executive function decline as well as basal forebrain and hippocampus atrophy in participants of the memory clinic based DELCODE cohort, spanning the range from cognitively normal through subjective cognitive decline, mild cognitive impairment and AD dementia. We had 794 cases with neuropsychological outcomes, and a subset of 703 cases with MRI outcomes. Effects were assessed using mixed effect models in a Bayesian framework using prior-insensitive cross-validated Bayes factors (CV-BF) and parameter estimates. Results: We found moderate evidence for an association of anticholinergic burden with baseline levels of cognitive impairment for the PACC5 as a global cognitive function score (CV-BF = 9.0) with more impairments with higher burden, but not with basal forebrain and hippocampus volumes, and weak evidence for an association of anticholinergic burden with longitudinal rates of change in the trailmaking test B as an executive function score (CV-BF = 2.5), but not for other cognitive scores and not for brain volumes. Conclusion: In the presence of prodromal or manifest AD, in a memory clinic-based cohort anticholinergic burden had only a modest effect on cognitive decline and no effect on atrophy in brain regions that are related to the cholinergic system.

Summary

Keywords

Alzheimer's disease, Cholinergic basal forebrain, Hippocampus, MRI, Treatement

Received

21 November 2025

Accepted

10 February 2026

Copyright

© 2026 Teipel, Grazia, Peters, Priller, Schneider, Wiltfang, Bartels, Schott, Jessen, Duzel, Yakupov, Buerger, Perneczky, Laske, Spottke, Wagner, Peltner, Kilimann and Haenisch. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Stefan Teipel; Alice Grazia

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