In the original article, there was a mistake in Figure 4 as published. The picture of “Control” in Figure 4C was wrong when we upload the pictures. The corrected Figure 4 appears below.
FIGURE 4
EFNB2 downregulation promotes the migration and invasion of HTR-8/SVneo cells. (A) EFNB2 mRNA expression examined after transfection of HTR-8/SVneo cells with siRNA for EFNB2. (B) Levels of EFNB2 and EMT-related proteins detected after 48-h transfection. (C,D) Transwell migration and invasion assays showing higher numbers of migrated and invaded cells in the si-EFNB2 group than in the NC group (×200). All data are presented as the mean ± standard deviation. EFNB2, Ephrin-B2; Si, siRNA; NC, negative control; EMT, epithelial-mesenchymal transition. *P < 0.05, **P < 0.01, ***P < 0.001, and ****P < 0.0001.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Statements
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
miR-193a-3p, EFNB2, epithelial-mesenchymal transition, decidua defect, placenta accreta spectrum
Citation
Li N, Hou R, Yang T, Liu C and Wei J (2022) Corrigendum: miR-193a-3p Mediates Placenta Accreta Spectrum Development by Targeting EFNB2 via Epithelial-Mesenchymal Transition Pathway Under Decidua Defect Conditions. Front. Mol. Biosci. 9:839235. doi: 10.3389/fmolb.2022.839235
Received
19 December 2021
Accepted
24 January 2022
Published
15 February 2022
Volume
9 - 2022
Edited and reviewed by
Yasuhito Ishigaki, Kanazawa Medical University, Japan
Updates
Copyright
© 2022 Li, Hou, Yang, Liu and Wei.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Caixia Liu, liucxshimen@163.com; Jun Wei, weij@sj-hospital.org
This article was submitted to Cellular Biochemistry, a section of the journal Frontiers in Molecular Biosciences
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.