In the published article, there was an error in affiliations [1, 2]. Instead of
“1Department of General Surgery, Institute for Minimally Invasive Surgery, Affiliated ZhongDa Hospital, Medical School, Southeast University, Dhaka, Bangladesh
2Department of Endcrinology, Affiliated ZhongDa Hospital, Medical School, Southeast University, Dhaka, Bangladesh, it should be
“1Department of General Surgery, Institute for Minimally Invasive Surgery, Affiliated ZhongDa Hospital, Medical School, Southeast University, Nanjing, China
2Department of Endcrinology, Affiliated ZhongDa Hospital, Medical School, Southeast University, Nanjing, China”
In the published article, there was an error regarding the affiliation(s) for Delin Liu. As well as having affiliation “1”, they should also have “Department of Endcrinology, Affiliated Zhongda Hospital, Medical School, Southeast University, Nanjing, China”.
A correction has been made to Results, The endothelial cells of keloid are divided into four subgroups, [176–180]. This sentence previously stated:
“genes upregulated in Endo1 were mainly involved in Osteoclast differentiation, TNF signaling Endo1 upregulated genes were mainly involved in Osteoclast differentiation, TNF signaling pathway, Apoptosis; Endo2 upregulated genes were mainly involved in IL-17 signaling pathway, Fluid shear stress and atherosclerosis, Rheumatoid arthritis; Endo3 upregulated genes were mainly involved in Endo3 upregulated genes are mainly involved in Endocrine resistance, Estrogen signaling pathway, Proteoglycans in cancer and other signaling pathways”
The corrected sentence appears below:
“genes upregulated in Endo1 were mainly involved in Osteoclast differentiation, TNF signaling pathway, Apoptosis; Endo2 upregulated genes were mainly involved in IL-17 signaling pathway, Fluid shear stress and atherosclerosis, Rheumatoid arthritis; Endo3 upregulated genes were mainly involved in Endocrine resistance, Estrogen signaling pathway, Proteoglycans in cancer and other signaling pathways”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Statements
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
keloid, IL6, endothelial cells (ECs), scRNAseq, NFkB (RelA)
Citation
Liu D, Zhang Y, Zhen L, Xu R, Ji Z and Ye Z (2022) Corrigendum: Activation of the NFκB signaling pathway in IL6+CSF3+ vascular endothelial cells promotes the formation of keloids. Front. Bioeng. Biotechnol. 10:1045496. doi: 10.3389/fbioe.2022.1045496
Received
15 September 2022
Accepted
20 September 2022
Published
02 November 2022
Approved by
Frontiers Editorial Office, Frontiers Media SA, Switzerland
Volume
10 - 2022
Updates
Copyright
© 2022 Liu, Zhang, Zhen, Xu, Ji and Ye.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Zhenling Ji, zhenlingji1@163.com; Zheng Ye, charles_ye@126.com
This article was submitted to Tissue Engineering and Regenerative Medicine, a section of the journal Frontiers in Bioengineering and Biotechnology
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.