The Publisher retracts the cited article.
Following publication, the publisher uncovered evidence that false identities were used in the peer-review process. The assignment of a fake reviewer was confirmed by an investigation, conducted in accordance with Frontiers’ policies and the Committee on Publication Ethics (COPE) guidelines.
The investigation also uncovered concerns about the presentation and validity of the data in the article that normally would have led to a rejection. When contacted, the authors failed to provide a data set that adequately supports the reported conclusions.
The authors do not agree to this retraction.
This retraction was approved by the Chief Editors of Frontiers in Cell and Developmental Biology and the Chief Executive Editor of Frontiers.
Summary
Keywords
cardio-cerebrovascult ischemia-reperfusion (I/R) injury, mitofusion 2 (Mfn2), Apoptosis, AMPK/Sirt3 signaling pathway, mitochondrial fusion
Citation
Frontiers Editorial Office (2022) Retraction: Mfn2 overexpression attenuates Cardio-Cerebrovascular Ischemia–Reperfusion injury through mitochondrial fusion and activation of the AMPK/Sirt3 signaling. Front. Cell Dev. Biol. 10:1006496. doi: 10.3389/fcell.2022.1006496
Received
29 July 2022
Accepted
02 August 2022
Published
04 August 2022
Approved by
Amanda Gay Fisher, Medical Research Council, United Kingdom
Volume
10 - 2022
Updates
Copyright
© 2022 Frontiers Editorial Office.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Frontiers Editorial Office, editorial.office@frontiersin.org
This article was submitted to Mitochondrial Research, a section of the journal Frontiers in Cell and Developmental Biology
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