In the published article, there was an error in Figure 2 as published. The photograph of U2OS N-cadherin WB strip in Figure 2B, the 143B miR-766-3p sh#2 cell migration and invasion photograph of Figures 2E, G are wrong. The corrected Figure 2 and its caption appear below.
FIGURE 2
Downregulating miR-766-3p promoted OS cell EMT, migration and invasion in vitro. (A) miR-766-3p lentiviruses were successfully transfected into 143B and U2OS cell lines (n = 3). (B) miR-766-3p sh#1 and miR-766-3p sh#2 increased the expression level of metastasis-related proteins in 143B and U2OS (n = 3). (C–F) The knockdown of miR-766-3p notably promoted the invasion and migration of 143B and U2OS cells (n = 4). (G, H) The Transwell invasion assays indicated that the knockdown of miR-766-3p significantly increased the invasive ability of OS cells (n = 4). (I–K) Colony formation, CCK-8 and EdU assays demonstrated that downregulating miR-766-3p promoted the proliferation of OS cells (n = 4). Data are presented as the means ± SD. *p < 0.01.
The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Statements
Publisher’s note
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Summary
Keywords
osteosarcoma, miR-766-3p, BCL9L, β-catenin, EMT, metastasis
Citation
Zhang S, Chen H, Liu W, Fang L, Qian Z, Kong R, Zhang Q, Li J and Cao X (2023) Corrigendum: miR-766-3p targeting BCL9L suppressed tumorigenesis, epithelial-mesenchymal transition, and metastasis through the β-catenin signaling pathway in osteosarcoma cells. Front. Cell Dev. Biol. 11:1239836. doi: 10.3389/fcell.2023.1239836
Received
14 June 2023
Accepted
27 October 2023
Published
15 November 2023
Volume
11 - 2023
Edited and reviewed by
Maurizio Onisto, University of Padua, Italy
Updates
Copyright
© 2023 Zhang, Chen, Liu, Fang, Qian, Kong, Zhang, Li and Cao.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Juming Li, lijuming@jsph.org.cn; Xiaojian Cao, xiaojiancao001@163.com
†These authors have contributed equally to this work
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.