In the original article, there was an error. The last paragraph contained several mistakes carried on from a previous version.
A correction has been made to the section Cardiac FGF23 Research: The Next Level, paragraph 2 and should read:
“Prior to any therapeutic intervention with the aim to minimize potentially negative FGF23 effects upon cardiac structure and function, research needs to focus on and clarify relevant unsolved issues. Just to name a few, the community needs to prove how cardiac disease induces (rather than follows) FGF-23 secretion, to what degree cardiomyocytes may themselves produce FGF-23 in health and disease, whether such locally produced FGF-23 has a physiological role in (acute) myocardial damage; and whether or not (systemic) FGF23 excess itself directly drives the development of myocardial damage. Only when these questions have been answered, can we try to discuss whether and how to intervene on serum FGF-23 level.”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way.
The original article has been updated.
Statements
Conflict of interest
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Summary
Keywords
FGF23, cardiovascular diseases, heart failure, hypertrophy, left ventricular, myocardial infarction
Citation
Stöhr R, Schuh A, Heine GH and Brandenburg V (2018) Corrigendum: FGF23 in Cardiovascular Disease: Innocent Bystander or Active Mediator?. Front. Endocrinol. 9:422. doi: 10.3389/fendo.2018.00422
Received
29 June 2018
Accepted
05 July 2018
Published
18 July 2018
Approved by
Frontiers in Endocrinology Editorial Office, Frontiers Media SA, Switzerland
Volume
9 - 2018
Updates
Copyright
© 2018 Stöhr, Schuh, Heine and Brandenburg.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Robert Stöhr rstoehr@ukaachen.de
This article was submitted to Molecular and Structural Endocrinology, a section of the journal Frontiers in Endocrinology
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