1. The Institute of Life Sciences, Engineering Laboratory of Zhejiang Province for Pharmaceutical Development of Growth Factors, Biomedical Collaborative Innovation Center of Wenzhou, Wenzhou University, Wenzhou, China
2. Research Units of Clinical Translation of Cell Growth Factors and Diseases Research of Chinese Academy of Medical Science, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, China
3. Clinical Research Center, Affiate Xiangshang Hospital, Wenzhou Medical University, Wenzhou, China
In the original article, there were some mistakes in “Figure 1G, (Figures 2A, C, and D) and Figure 6B” as published. The image of representative swimming track of db/m mice in the probe trial (24 h after training) (Figure 1G), and the images of immunohistochemical staining of Aβ1−42 and immunofluorescence staining of p-Tau in the CA1 region of db/m mice (Figure 2A) should be replaced with appropriate images. The last result of quantitative analysis in Figure 6B should be the quantitative analysis of CHOP. The labels in Figures 2C and D of Synapthysin-1 and Synapsin should be Synapthysin and Synapsin-1. The corrected Figures appears below.
Figure 1
Figure 2
Figure 6
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Wu Y, Yuan Y, Wu C, Jiang T, Wang B, Xiong J, Zheng P, Li Y, Xu J, Xu K, Liu Y, Li X and Xiao J (2021) Corrigendum: The Reciprocal Causation of the ASK1-JNK1/2 Pathway and Endoplasmic Reticulum Stress in Diabetes-Induced Cognitive Decline. Front. Cell Dev. Biol. 9:639486. doi: 10.3389/fcell.2021.639486
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†These authors have contributed equally to this work
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