CORRECTION article

Front. Cell Dev. Biol.

Sec. Cell Adhesion and Migration

Volume 13 - 2025 | doi: 10.3389/fcell.2025.1605628

Corrigendum: Loss of CDYL Results in Suppression of CTNNB1 and Decreased Endometrial Receptivity

Provisionally accepted
  • 1Department of Reproductive Medical Center, Ruijin Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China
  • 2Department of Obstetrics and Gynaecology, Chinese People's Armed Police Force Shanghai Corps Hospital, Shanghai, China
  • 3Department of Obstetrics and Gynaecology, BC Children's Hospital Research Institute, Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada
  • 4Department of Histoembryology, Genetics and Developmental Biology, School of Medicine, Shanghai Jiao Tong University, Shanghai, Shanghai Municipality, China
  • 5Shanghai Key Laboratory of Reproduction Medicine, School of Medicine, Shanghai Jiao Tong University, Shanghai, China

The final, formatted version of the article will be published soon.

In the published article, there was an error in Figure 3C as published. The authors have identified that the CDYL-sh1 48h treatment was duplicated from CDYL-sh3 24h treatment due to an error during figure preparation. The corrected Figure 3C and its caption CDYL knockdown suppressed the cell migration capability in Ishikawa cells appear below. In the published article, there was an error in Figure 5F as published. The Western blot for CDYL in Figure 5F, while from the same experimental day, was not from the same membrane as CTNNB1 and GAPDH, due to an error during figure preparation. The corrected Figure 5F and its caption the decreased expression of CTNNB1 after CDYL knockdown in Ishikawa cells, EECs, and ESCs appear below.The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Keywords: recurrent implantation failure, chromodomain Y like, catenin beta 1, Migration, Endometrial receptivity

Received: 03 Apr 2025; Accepted: 16 Jun 2025.

Copyright: © 2025 Zhou, Xu, Zhang, Jiang, Chang, Leung, Xia and Zhang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

* Correspondence:
Xiaowei Zhou, Department of Reproductive Medical Center, Ruijin Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China
Xiaoyu Xia, Department of Histoembryology, Genetics and Developmental Biology, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200025, Shanghai Municipality, China
Aijun Zhang, Department of Reproductive Medical Center, Ruijin Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China

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