CORRECTION article

Front. Immunol., 05 August 2025

Sec. Microbial Immunology

Volume 16 - 2025 | https://doi.org/10.3389/fimmu.2025.1643508

Correction: Exosomal let-7b-5p derived from Aspergillus fumigatus-treated human corneal epithelial cells promotes M1 macrophage activation via targeting SOCS-1

  • 1. Department of Ophthalmology, Qilu Hospital of Shandong University, No.107, Wenhua Xilu, Jinan, Shandong, China

  • 2. Department of Ophthalmology, Shandong Provincial Third Hospital, Shandong University, Jinan, Shandong, China

There was a mistake in Figure 1 as published. Figure 1A, title changed to HEXPBS (PBS superscript); Figure 1C, first target protein labelled as Calnexin. The corrected Figure 1 appears below.

Figure 1

The original version of this article has been updated.

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Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

fungal keratitis, exosomes, let-7b-5p, SOCS-1, M1 macrophage activation

Citation

Yu X and Wu X (2025) Correction: Exosomal let-7b-5p derived from Aspergillus fumigatus-treated human corneal epithelial cells promotes M1 macrophage activation via targeting SOCS-1. Front. Immunol. 16:1643508. doi: 10.3389/fimmu.2025.1643508

Received

09 June 2025

Accepted

01 July 2025

Published

05 August 2025

Volume

16 - 2025

Edited and reviewed by

Fausto Almeida, University of São Paulo, Brazil

Updates

Copyright

*Correspondence: Xinyi Wu,

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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