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CORRECTION article

Front. Immunol., 08 October 2025

Sec. Autoimmune and Autoinflammatory Disorders : Autoimmune Disorders

Volume 16 - 2025 | https://doi.org/10.3389/fimmu.2025.1711668

This article is part of the Research TopicBiologics and Targeted Therapies for Autoimmune and Auto-inflammatory Dermatoses: Balancing Efficacy with Safety and ToxicityView all 5 articles

Correction: Novel advances on pathophysiological mechanisms, clinical manifestations, and treatment of antiphospholipid syndrome

Qing-Nan Zhu,Qing-Nan Zhu1,2Xiang-Bo QiXiang-Bo Qi2Shu-Wei RenShu-Wei Ren2Yu-Ye LiYu-Ye Li2Ze-Wen YanZe-Wen Yan2Yu SunYu Sun2Yan ShiYan Shi2Qing-Si WenQing-Si Wen2Mao-Mao WuMao-Mao Wu2Da-Peng Wang*Da-Peng Wang1*
  • 1Department of Nephrology, The First Affiliated Hospital, Dalian Medical University, Dalian, China
  • 2Institute of Integrative Medicine, Dalian Medical University, Dalian, China

A Correction on
Novel advances on pathophysiological mechanisms, clinical manifestations, and treatment of antiphospholipid syndrome

By Zhu Q-N, Qi X-B, Ren S-W, Li Y-Y, Yan Z-W, Sun Y, Shi Y, Wen Q-S, Wu M-M and Wang D-P (2025). Front. Immunol. 16:1639065. doi: 10.3389/fimmu.2025.1639065

Affiliation “Department of Nephrology, The First Affiliated Hospital, Dalian Medical University, Dalian, China” was omitted for author Qing-Nan Zhu. This affiliation has now been added for author Qing-Nan Zhu. As such, affiliation “Institute of Integrative Medicine, Dalian Medical University, Dalian, China” previously listed as affiliation 1 is now affiliation 2 and the author list has been updated accordingly.

In the published article, there was a mistake in the Figure order. The image labeled as “Figure 1” should be “Figure 2” and the image labeled as “Figure 2” should be “Figure 1”. The order has now been corrected.

Figure 1
Diagram illustrating the impact of platelet aggregation on fibrinolysis and cellular activation. It contrasts physiological status with impaired fibrinolysis. The left side shows platelets, neutrophils, monocytes, and lymphocytes in normal states. The right side reveals activated platelets and cells with pathways involving aPLs, β2GPI, TLRs, and other molecules leading to increased cytokines and complement activation. Processes like NETosis and adhesion molecule expression are depicted.

Figure 1. Pathogenesis of antiphospholipid syndrome (APS): cellular mechanisms.

Figure 2
Diagram outlining various manifestations of Antiphospholipid Syndrome (APS) on the human body. Key areas affected include ocular (retinal artery/vein occlusion), cardiac (myocardial infarction, coronary artery disease), neurological (cognitive dysfunction, seizures), pulmonary (embolism), renal (aPL-associated nephropathy), cutaneous (livedo reticularis), obstetric (recurrent pregnancy loss), hematological (thrombocytopenia), and vessels (deep vein thrombosis). Each system is connected to the central anatomical depiction of the human body.

Figure 2. Manifestation of APS.

The original version of this article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: antiphospholipid syndrome, systemic lupus erythematosus, thromboinflammation, thrombosis, anticoagulants

Citation: Zhu Q-N, Qi X-B, Ren S-W, Li Y-Y, Yan Z-W, Sun Y, Shi Y, Wen Q-S, Wu M-M and Wang D-P (2025) Correction: Novel advances on pathophysiological mechanisms, clinical manifestations, and treatment of antiphospholipid syndrome. Front. Immunol. 16:1711668. doi: 10.3389/fimmu.2025.1711668

Received: 23 September 2025; Accepted: 03 October 2025;
Published: 08 October 2025.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2025 Zhu, Qi, Ren, Li, Yan, Sun, Shi, Wen, Wu and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Da-Peng Wang, Ym9ib3dkcEAxNjMuY29t

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.