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Autophagy in Autoimmunity

Correction ARTICLE

Front. Immunol., 16 November 2018 | https://doi.org/10.3389/fimmu.2018.02627

Corrigendum: Lymphocyte Autophagy in Homeostasis, Activation, and Inflammatory Diseases

  • 1CNRS UPR3572, Immunology, Immunopathology and Therapeutic Chemistry/Laboratory of Excellence MEDALIS, Institut de Biologie Moléculaire et Cellulaire, Strasbourg, France
  • 2University of Strasbourg, Strasbourg, France

A Corrigendum on
Lymphocyte Autophagy in Homeostasis, Activation, and Inflammatory Diseases

by Arbogast, F., and Gros, F. (2018). Front. Immunol. 9:1801. doi: 10.3389/fimmu.2018.01801

In the original article, two clarifications about cited references are necessary.

First, the sentence “As a consequence, autophagy-deficient T cells show impaired TH9 differentiation and antitumor responses (63)” should be “As a consequence, autophagy-deficient T cells show enhanced TH9-dependent anti-tumor responses (63)”. A correction has been made to the section Autophagy in Peripheral T Cells, Macroautophagy in T Cell Activation, paragraph 2.

Moreover, even if mechanisms are not totally understood, Chen et al. indeed found experimental evidence in [42], for a role played by autophagy in limiting lipid peroxidation toxicity induced by reactive oxygen species. The sentence “To date, no mechanism linking autophagy and memory B cell survival has been proposed. It is possible that mitophagy and mobilization of lipids through lipophagy might be important, as for T cells” has been corrected to “Chen et al. (42) showed that autophagy in memory B cells limits mitochondrial ROS production and toxicity of peroxidized lipids. It is also possible that mobilization of lipids through lipophagy might be required for the survival of both memory B and T cells”. A correction has been made to the section Autophagy in peripheral B Cells, Macroautophagy in Memory B Cell and Plasma Cell Survival, paragraph 2.

The authors apologize for these errors and state that they do not change the scientific conclusions of the article in any way. The original article has been updated.

Conflict of Interest Statement

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

References

42. Chen M, Hong MJ, Sun H, Wang L, Shi X, Gilbert BE, et al. Essential role for autophagy in the maintenance of immunological memory against influenza infection. Nat Med. (2014) 20:503–10. doi: 10.1038/nm.3521

PubMed Abstract | CrossRef Full Text | Google Scholar

63. Rivera Vargas T, Cai Z, Shen Y, Dosset M, Benoit-Lizon I, Martin T, et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. (2017) 8:559. doi: 10.1038/s41467-017-00468-w

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Keywords: autophagy, mitophagy, metabolism, unfolded protein response, autoimmunity, lymphocytes

Citation: Arbogast F and Gros F (2018) Corrigendum: Lymphocyte Autophagy in Homeostasis, Activation, and Inflammatory Diseases. Front. Immunol. 9:2627. doi: 10.3389/fimmu.2018.02627

Received: 24 August 2018; Accepted: 25 October 2018;
Published: 16 November 2018.

Edited and reviewed by: Marko Radic, University of Tennessee College of Medicine, United States

Copyright © 2018 Arbogast and Gros. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Frédéric Gros, f.gros@ibmc-cnrs.unistra.fr