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CORRECTION article

Front. Immunol., 22 May 2024
Sec. Molecular Innate Immunity

Corrigendum: Expression of constitutive fusion of ubiquitin to PCNA restores the level of immunoglobulin A/T mutations during somatic hypermutation in the Ramos cell line

Leticia K. Lerner,,,&#x;&#x;Leticia K. Lerner1,2,3,4†‡Dorine Bonte,,&#x;Dorine Bonte1,2,3‡Morwenna Le Guillou,,Morwenna Le Guillou1,2,3Mahwish Mian Mohammad,,,Mahwish Mian Mohammad1,2,3,5Zeinab Kasraian,,Zeinab Kasraian1,2,3Alain Sarasin,,Alain Sarasin1,2,3Emmanuelle Despras,,&#x;Emmanuelle Despras1,2,3†Said Aoufouchi,,,*Said Aoufouchi1,2,3,5*
  • 1Centre National de la Recherche Scientifique UMR 9019, B Cell and Genome Plasticity Team, Villejuif, France
  • 2Gustave Roussy, Villejuif, France
  • 3Université Paris-Saclay, Orsay, France
  • 4Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil
  • 5Sorbonne Université, Paris, France

A Corrigendum on
Expression of constitutive fusion of ubiquitin to PCNA restores the level of immunoglobulin A/T mutations during somatic hypermutation in the Ramos cell line

By Lerner LK, Bonte D, Le Guillou M, Mohammad MM, Kasraian Z, Sarasin A, Despras E and Aoufouchi S (2022). Front. Immunol. 13:871766. doi: 10.3389/fimmu.2022.871766

In the published article, there was an error in Figure 5 as published. The published version contains typing errors. The corrected Figure 5 and its caption appear below.

Figure 5
www.frontiersin.org

Figure 5 Distribution of point-mutations along the amplified Ramos VH region. Independently occurring base substitutions are indicated at each nucleotide position. The POLH hotspots (WA/TW) targeted following the expression of mUb-PCNA are in indicated in red. The figure represent the pool of base substitution obtained from the clones indicated in Table 2A. The Nucleotide Substitutions in blue indicated above the Ramos VH sequence are from the 5 clones expressing mUb-PCNA and those below in green are from the five control clones.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: immunoglobulin somatic hypermutation, PCNA monoubiquitination, Ramos B cell line, USP1 inhibition, A/T mutation pathway

Citation: Lerner LK, Bonte D, Le Guillou M, Mohammad MM, Kasraian Z, Sarasin A, Despras E and Aoufouchi S (2024) Corrigendum: Expression of constitutive fusion of ubiquitin to PCNA restores the level of immunoglobulin A/T mutations during somatic hypermutation in the Ramos cell line. Front. Immunol. 15:1419493. doi: 10.3389/fimmu.2024.1419493

Received: 18 April 2024; Accepted: 14 May 2024;
Published: 22 May 2024.

Edited and Reviewed by:

Francesca Granucci, University of Milano-Bicocca, Italy

Copyright © 2024 Lerner, Bonte, Le Guillou, Mohammad, Kasraian, Sarasin, Despras and Aoufouchi. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Said Aoufouchi, said.aoufouchi@gustaveroussy.fr

Present address: Leticia K. Lerner, Cell Death and Drug Resistance in Hematological Disorders Team, Centre de Recherche des Cordeliers, INSERM, Sorbonne Université, Université de Paris, Paris, France Emmanuelle Despras, Sorbonne Université, INSERM UMRS 938, Équipe Instabilité des Microsatellites et Cancer, Centre de recherche Saint Antoine, Paris, France

These authors have contributed equally to this work and share first authorship

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.