CORRECTION article

Front. Immunol., 23 June 2025

Sec. Inflammation

Volume 16 - 2025 | https://doi.org/10.3389/fimmu.2025.1637511

Correction: Protein carbamylation in atherosclerotic plaques correlates with uremia and disease progression, localizing predominantly to foam cells

  • 1. Department of Pathology, Cardiovascular Research Institute Maastricht, Maastricht University Medical Center, Maastricht, Netherlands

  • 2. Department of Hepatology and Gastroenterology, Charité - Universitätsmedizin Berlin, Campus Virchow-Klinikum and Campus Charité Mitte, Berlin, Germany

  • 3. School for Oncology and Developmental Biology (GROW), Maastricht University Medical Center, Maastricht, Netherlands

  • 4. Department of Medical Biochemistry, Experimental Vascular Biology, Amsterdam University Medical Centers, University of Amsterdam, Amsterdam, Netherlands

  • 5. Centre for Cardiovascular Science, University of Edinburgh, Edinburgh, United Kingdom

  • 6. Department of Surgery, Maastricht University Medical Center (UMC)+, Maastricht, Netherlands

  • 7. Institute for Molecular Cardiovascular Research (IMCAR), Rheinisch-Westfälische Technische Hochschule (RWTH) Aachen University, Aachen, Germany

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In the published article, there was an error in the Funding statement. The funding mentioned “This project has received funding from the European Union’s Horizon 2020 research and innovation programme under the Marie Skłodowska-Curie grant agreement No 764474 (CaReSyAn).” was incomplete. The correct Funding statement appears below.

“This project has received funding from the European Union’s Horizon 2020 research and innovation programme under the Marie Skłodowska-Curie grant agreement No 764474 (CaReSyAn). VJankowski and JJankowski are funded by the German Research Foundation (DFG) through the Transregional Collaborative Research Center (TRR 219; Project ID 322900939), (subproject S-03), (INST 948/4S-1); CRU 5011 project number 445703531, Cost-Action CA 21165, IZKF Multiorgan complexity in Friedreich Ataxia, and Phase Transition in Disease 1-1), ERA-PerMed (ERA-PERMED2022-202-KidneySign).”

The original article has been updated.

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Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

atherosclerosis, macrophages, foam cells, carbamylation, kidney disease

Citation

Saar-Kovrov V, Pawlowska A, Guillot A, Gijbels MJJ, Sluimer JC, Temmerman L, Goossens P, Mees BME, Tacke F, Jankowski V, Jankowski J, Donners MMPC and Biessen EAL (2025) Correction: Protein carbamylation in atherosclerotic plaques correlates with uremia and disease progression, localizing predominantly to foam cells. Front. Immunol. 16:1637511. doi: 10.3389/fimmu.2025.1637511

Received

29 May 2025

Accepted

05 June 2025

Published

23 June 2025

Approved by

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Volume

16 - 2025

Updates

Copyright

*Correspondence: Erik A. L. Biessen,

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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