ORIGINAL RESEARCH article
Front. Pharmacol., 25 March 2019
Sec. Renal Pharmacology
Volume 10 - 2019 | https://doi.org/10.3389/fphar.2019.00224
RETRACTED: CXCL6 Promotes Renal Interstitial Fibrosis in Diabetic Nephropathy by Activating JAK/STAT3 Signaling Pathway
- MS
Meng-Yao Sun 1,2†
- SW
Su-Juan Wang 1,3†
- XL
Xiao-Qin Li 1
- YS
Yu-Li Shen 1
- JL
Jian-Rao Lu 4
- XT
Xin-Hui Tian 1
- KR
Khalid Rahman 5
- LZ
Li-Jun Zhang 1*
- HN
Hua Nian 6*
- HZ
Hong Zhang 1*
1. Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China
2. Central Laboratory, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China
3. Department of Drug Preparation, Hospital of TCM and Hui Nationality Medicine, Ningxia Medical University, Wuzhong, China
4. Department of Nephrology, Seventh People’s Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China
5. School of Pharmacy and Biomolecular Sciences, Faculty of Science, Liverpool John Moores University, Liverpool, United Kingdom
6. Department of Pharmacy, Yueyang Hospital of Integrated Traditional Chinese and Western Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, China
Summary
Citation
Sun M-Y, Wang S-J, Li X-Q, Shen Y-L, Lu J-R, Tian X-H, Rahman K, Zhang L-J, Nian H and Zhang H (2019) RETRACTED: CXCL6 Promotes Renal Interstitial Fibrosis in Diabetic Nephropathy by Activating JAK/STAT3 Signaling Pathway. Front. Pharmacol. 10:224. doi: 10.3389/fphar.2019.00224
Received
25 September 2018
Accepted
22 February 2019
Published
25 March 2019
Volume
10 - 2019
Edited by
Matthew Griffin, National University of Ireland Galway, Ireland
Reviewed by
Peter Jon Nelson, Ludwig Maximilian University of Munich, Germany; Ana Belen Sanz, Instituto de Investigación Sanitaria Fundación Jiménez Díaz, Spain
Updates
†These authors have contributed equally to this work
This article was submitted to Renal Pharmacology, a section of the journal Frontiers in Pharmacology
Disclaimer
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