CORRECTION article

Front. Cell. Infect. Microbiol., 29 July 2022

Sec. Virus and Host

Volume 12 - 2022 | https://doi.org/10.3389/fcimb.2022.964880

Corrigendum: brain microvascular endothelial cell-derived HMGB1 facilitates monocyte adhesion and transmigration to promote JEV neuroinvasion

  • SZ

    Song-Song Zou 1,2,3,4

  • QZ

    Qing-Cui Zou 1,2,3,4

  • WX

    Wen-Jing Xiong 1,2,3,4

  • NC

    Ning-Yi Cui 1,2,3,4

  • KW

    Ke Wang 1,2,3,4

  • HL

    Hao-Xuan Liu 1,2,3,4

  • WL

    Wen-Juan Lou 1,2,3,4

  • DH

    Doaa Higazy 1,2,3,4

  • YZ

    Ya-Ge Zhang 1,2,3,4

  • MC

    Min Cui 1,2,3,4*

  • 1. State Key Laboratory of Agricultural Microbiology, College of Veterinary Medicine, Huazhong Agricultural University, Wuhan, China

  • 2. Key Laboratory of Preventive Veterinary Medicine in Hubei Province, The Cooperative Innovation Center for Sustainable Pig Production, Wuhan, China

  • 3. Key Laboratory of Development of Veterinary Diagnostic Products, Ministry of Agriculture of the People’s Republic of China, Wuhan, China

  • 4. International Research Center for Animal Disease, Ministry of Science and Technology of the People’s Republic of China, Wuhan, China

In the original article, there was a mistake in Figures 5D–E as published. Images repeated ofFigures 5D–E. The corrected Figure 5 appears below.

Figure 5

In the original article, there was an error in the description ofFigure 5D.

A correction has been made to Results, Extracellular HMGB1 Facilitated Transendothelial Migration of JEV-Infected Monocytes, **paragraph 3:

To discover which cells act as virus carriers, JEV with an EGFP tag (EGFP-JEV) was applied to visualize cell transmigration. There was an increased percentage of EGFP-positive Ly6C+CD11b+ monocytes, CD3+ T cells, and CD19+ B cells that transmigrated, compared with the control cells (Figures 5A–C). Furthermore, there were significantly more transmigrated JEV-positive (EGFP+Ly6C+CD11b+) monocytes than transmigrated JEV-positive T cells (EGFP+CD3+) or B cells (EGFP+CD19+) (Figures 5D, E).

The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s Note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Statements

Conflict of interest

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

Summary

Keywords

transmigration, adhesion, monocyte, HMGB1, Japanese encephalitis virus (JEV), neuroinvasion

Citation

Zou S-S, Zou Q-C, Xiong W-J, Cui N-Y, Wang K, Liu H-X, Lou W-J, Higazy D, Zhang Y-G and Cui M (2022) Corrigendum: brain microvascular endothelial cell-derived HMGB1 facilitates monocyte adhesion and transmigration to promote JEV neuroinvasion. Front. Cell. Infect. Microbiol. 12:964880. doi: 10.3389/fcimb.2022.964880

Received

09 June 2022

Accepted

14 July 2022

Published

29 July 2022

Volume

12 - 2022

Edited and reviewed by

Abrar Hussain, Balochistan University of Information Technology, Engineering and Management Sciences, Pakistan

Updates

Copyright

*Correspondence: Min Cui,

This article was submitted to Virus and Host, a section of the journal Frontiers in Cellular and Infection Microbiology

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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