CORRECTION article

Front. Endocrinol., 26 June 2023

Sec. Cellular Endocrinology

Volume 14 - 2023 | https://doi.org/10.3389/fendo.2023.1219036

Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism

  • 1. Doctoral School of Molecular Cell and Immunobiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary

  • 2. Department of Biochemistry and Molecular Biology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary

  • 3. Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, TN, United States

  • 4. Immunometabolism Research Group, Department of Cell and Developmental Biology, University of São Paulo, São Paulo, Brazil

  • 5. Department of Medicine, Johns Hopkins University School of Medicine, and Institute for Fundamental Biomedical Research, Johns Hopkins All Children’s Hospital, St Petersburg, FL, United States

  • 6. Department of Biological Chemistry, Johns Hopkins University School of Medicine, and Institute for Fundamental Biomedical Research, Johns Hopkins All Children’s Hospital, St Petersburg, FL, United States

In the published article, Figure 1 and the respective citations were omitted. The corrected Figure 1 and its caption appear below.

Figure 1

The citations for Figure 1 have now been inserted in paragraphs 3 and 4 and should read:

“In a recently published paper, Merz et al. link the relevance of PAK1 to type II diabetes pathogenesis by the use of knockout and overexpression of PAK1 mouse models (Figure 1).”

“Nonetheless, the interconnection between insulin release, resistance, and glucose metabolism in skeletal muscle is much more complex and it also has an immunological cofactor as reviewed by Shen et al. linking the systematic pro-inflammatory response detected by several studies by the increased levels of TNFa or IL-6 in serum (8) and intramuscular (9) with muscle degeneration (Figure 1).”

“In this issue, Arias-Calderoín et al. have been able to identify that FGF21 an important hormone for muscle repair, can be secreted intra-muscularly via PI3K/AKT/mTOR signaling pathway (Figure 1).”

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Statements

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

skeletal muscle, glucose metabolism, tissue cross talk, inflammation, FGF21

Citation

Caballero-Sánchez N, Winn N, Rosa Neto JC and Nagy L (2023) Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism. Front. Endocrinol. 14:1219036. doi: 10.3389/fendo.2023.1219036

Received

08 May 2023

Accepted

08 June 2023

Published

26 June 2023

Approved by

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Volume

14 - 2023

Updates

Copyright

*Correspondence: Laszlo Nagy,

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

Outline

Figures

Cite article

Copy to clipboard


Export citation file


Share article

Article metrics