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CORRECTION article

Front. Endocrinol., 19 January 2026

Sec. Endocrinology of Aging

Volume 16 - 2025 | https://doi.org/10.3389/fendo.2025.1748130

Correction: Remnant cholesterol shows inverse and nonlinear associations with leukocyte telomere length and serum α-Klotho, mediated by inflammation and oxidative stress

  • 1Department of Endocrinology, Key Laboratory of Endocrinology of National Health Commission, Translation Medicine Center, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China
  • 2Department of Nutrition, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China

A Correction on
Remnant cholesterol shows inverse and nonlinear associations with leukocyte telomere length and serum α-Klotho, mediated by inflammation and oxidative stress

By Xing B, Yu J, Liu Y, Gao Q, Chen X, He S, Ping F, Xu L, Li W, Zhang H and Li Y (2025) Front. Endocrinol. 16:1700349. doi: 10.3389/fendo.2025.1700349

There was a mistake in Table 2 as published. The regression coefficients for serum α-Klotho of models 3 and 4 in Table 2 were wrong. The corrected Table 2 appears below.

Table 2
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Table 2. The multiple linear regression between RC and LTL and serum α-klotho.

There was a mistake in Figure 2c as published. There was a missing * for the total effect. The corrected Figure 2 appears below. The original version of this article has been updated.

Figure 2
Diagrams depicting the relationship between RC, various mediators, and LTL. (a) TNFα, (b) IL-6, (c) IL-1β, (d) SOD, and (e) 8-oHdG. Each has paths showing indirect, direct, and total effects with corresponding values and confidence intervals. Each mediator affects the relationship between RC and LTL differently, with asterisks indicating significance.

Figure 2. The mediation effect of inflammation and oxidative stress in the relationship between RC and LTL. (a–e) the mediation model of TNFα, IL-6, IL-1β, SOD, 8-OHdG on the correlation between RC and LTL. The model was adjusted for age, sex, BMI, WHR, ALT, AST, eGFR, SBP, DBP, HbA1c, FPG, LDL-C, HDL-C, TG, sUA, and total energy intake. The z-LTL and log-transformed TNFα, IL-6, IL-1β, SOD, and 8-OHdG were analyzed in the model. *P>0.05 means statistical difference..

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: remnant cholesterol, biological aging, leukocyte telomere length, serum α-Klotho, inflammation, oxidative stress

Citation: Xing B, Yu J, Liu Y, Gao Q, Chen X, He S, Ping F, Xu L, Li W, Zhang H and Li Y (2026) Correction: Remnant cholesterol shows inverse and nonlinear associations with leukocyte telomere length and serum α-Klotho, mediated by inflammation and oxidative stress. Front. Endocrinol. 16:1748130. doi: 10.3389/fendo.2025.1748130

Received: 17 November 2025; Accepted: 29 December 2025;
Published: 19 January 2026.

Edited and reviewed by:

James Harper, Sam Houston State University, United States

Copyright © 2026 Xing, Yu, Liu, Gao, Chen, He, Ping, Xu, Li, Zhang and Li. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Huabing Zhang, aHVhYmluZ3poYW5nY2huQDE2My5jb20=; Yuxiu Li, bGl5dXhpdUBtZWRtYWlsLmNvbS5jbg==

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.