RETRACTION article

Front. Immunol., 14 June 2019

Sec. Antigen Presenting Cell Biology

Volume 10 - 2019 | https://doi.org/10.3389/fimmu.2019.01468

Retraction: Stimulator of Interferon Genes in Classical Dendritic Cells Controls Mucosal Th17 Responses to Cyclic Dinucleotides for Host Defenses Against Microbial Infections in Gut

  • Frontiers Media SA, Lausanne, Switzerland

The journal and Chief Editors retract the 16 May 2018 article cited above.

Following the publication of the article, concerns regarding the source of the data used in the article were addressed to the journal and its Editorial Board. Following clarification with the authors, it was identified that data generated at a different institute were partially included in their article erroneously. The article is therefore being retracted.

The authors concur with the retraction and sincerely regret any inconvenience this may have caused to the reviewers, editors, and readers of Frontiers in Immunology.

Summary

Keywords

STING, dendritic cell, Th17, Salmonella, cyclic dinucleotides (CDNs), Crohn ‘s disease

Citation

Frontiers Editorial Office (2019) Retraction: Stimulator of Interferon Genes in Classical Dendritic Cells Controls Mucosal Th17 Responses to Cyclic Dinucleotides for Host Defenses Against Microbial Infections in Gut. Front. Immunol. 10:1468. doi: 10.3389/fimmu.2019.01468

Received

11 June 2019

Accepted

11 June 2019

Published

14 June 2019

Approved by

Florent Ginhoux, Singapore Immunology Network (A*STAR), Singapore

Volume

10 - 2019

Updates

Copyright

*Correspondence: Frontiers Editorial Office

This article was submitted to Antigen Presenting Cell Biology, a section of the journal Frontiers in Immunology

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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