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CORRECTION article

Front. Immunol., 08 April 2024
Sec. Multiple Sclerosis and Neuroimmunology

Corrigendum: Characterization of pannexin1, connexin32, and connexin43 in spotted sea bass (Lateolabrax maculatus): they are important neuro-related immune response genes involved in inflammation-induced ATP release

  • 1Key Laboratory of Exploration and Utilization of Aquatic Genetic Resources, Ministry of Education, Shanghai Ocean University, Shanghai, China
  • 2International Research Center for Marine Biosciences at Shanghai Ocean University, Ministry of Science and Technology, Shanghai, China
  • 3National Demonstration Center for Experimental Fisheries Science Education, Shanghai Ocean University, Shanghai, China
  • 4College of Life Science and Technology, Huazhong University of Science and Technology, Wuhan, China

Error in Figure/Table

In the published article, there was an error in Figure 8 as published. In Figure 8A, we mistakenly used a figure from our previous publication. The corrected Figure 8 and its caption appear below.

Figure 8
www.frontiersin.org

Figure 8 Subcellular localization of LmPanx1, LmCx32, and LmCx43 in HEK293 T cells (A) and LPS-induced extracellular ATP release in primary head kidney leukocytes (B) or HEK293 T cells (C). (A) HEK293 T cells were transfected with pEGFP-N1-LmPanx1, pEGFP-N1 LmCx32.2, pEGFP-N1-LmCx32.3, pEGFP-N1-LmCx32.7, or pEGFP-N1-LmCx43 plasmids. At 24 h post transfection, the cells were stained with DAPI and observed under a confocal microscope. (B, C) The primary head kidney leukocytes were stimulated with LPS (100 µg/mL) or PBS (control). HEK293 T cells were transfected with pcDNA3.1-LmPanx1, pcDNA3.1-LmCx32.2, pcDNA3.1-LmCx32.3, pcDNA3.1-LmCx32.7, or pcDNA3.1-LmCx43. After 24 h, the cells were stimulated with LPS (100 µg/mL). The supernatant was collected at 15 and 30 min after stimulation and the ATP levels were subsequently measured. The mock transfected and empty plasmid transfected cells served as controls. Data are shown as mean +SEM (N = 4). *p < 0.05, **p < 0.01 are considered significant difference.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: pannexin1, connexin32, connexin43, innate immunity, ATP release, Lateolabrax maculates

Citation: Sun Z, Xu C, Chen Y, Liu D, Wu P and Gao Q (2024) Corrigendum: Characterization of pannexin1, connexin32, and connexin43 in spotted sea bass (Lateolabrax maculatus): they are important neuro-related immune response genes involved in inflammation-induced ATP release. Front. Immunol. 15:1377603. doi: 10.3389/fimmu.2024.1377603

Received: 05 February 2024; Accepted: 26 March 2024;
Published: 08 April 2024.

Edited and Reviewed by:

Robert Weissert, University of Regensburg, Germany

Copyright © 2024 Sun, Xu, Chen, Liu, Wu and Gao. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Qian Gao, cWdhb0BzaG91LmVkdS5jbg==; Ping Wu, d3UtcGluZ0B3aHUuZWR1LmNu

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.