In the published article, there was an error in Figure 2D as published. In the original Figure 2D, the Celastrol panels for HT-29 cell line were mistakenly placed. The corrected Figure 2D and its caption **HSP70 regulated anti-apoptosis, cell survival, and NO levels. (D) HSP70-mediated regulation of cleaved CASP-3 as examined using immunofluorescence assay (scale bar = 100 μm) appear below.
Figure 2
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Statements
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
Giardia, HSP70, host defense, apoptosis, nitric oxide, tight junction
Citation
Liu M, Yang Y, Zhu W, Wu J, Yu X and Li W (2024) Corrigendum: Specific TLR-mediated HSP70 activation plays a potential role in host defense against the intestinal parasite Giardia duodenalis. Front. Microbiol. 15:1359801. doi: 10.3389/fmicb.2024.1359801
Received
22 December 2023
Accepted
18 January 2024
Published
02 February 2024
Volume
15 - 2024
Edited and reviewed by
Axel Cloeckaert, Institut National de recherche pour l'agriculture, l'alimentation et l'environnement (INRAE), France
Updates
Copyright
© 2024 Liu, Yang, Zhu, Wu, Yu and Li.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Wei Li neaulw@gmail.com
†These authors have contributed equally to this work
‡ORCID: Wei Li orcid.org/0000-0002-4264-1864
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.