CORRECTION article

Front. Oncol.

Sec. Cancer Metabolism

Volume 15 - 2025 | doi: 10.3389/fonc.2025.1630154

Corrigendum: Rewired glycolysis by DTL accelerates oncometabolite L-lactate generation to promote breast cancer progression

Provisionally accepted
Mengzhu  LvMengzhu Lv1*YuHao  LiuYuHao Liu2Jinting  LiJinting Li1Yiren  CaoYiren Cao1*
  • 1Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Laboratory of Molecular Oncology, Peking University Cancer Hospital and Institute, Beijing, China, Beijing, China
  • 2Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Department of Radiation Oncology, Peking University Cancer Hospital and Institute, Beijing, China, Beijing, China

The final, formatted version of the article will be published soon.

• please read through all the templates before choosing • pick the most relevant text template(s) from the following page and delete all others.• edit the text as necessary, ensuring that the original incorrect text is included for the record, please see the below. • please do not use any extra formatting when editing the templates, and only modify the red text unless absolutely necessary • submit to Frontiers following the instructions on this page.When the original text contained incorrect information, to preserve the scientific record, please include that text when editing the below templates. For example:There was a mistake in the Funding statement, an incorrect number was used. The correct number is "2015C03Bd051.". The publisher apologizes for this mistake.The original version of this article has been updated. In the published article, there was an error in Figure 6C as published. We regrettably discovered an inadvertent duplication of loading control bands (β-actin and GAPDH for CAL-51 cell line) between Figure 5A and Figure 6C during figure reorganization. We wish to emphasize that the duplicated controls originated from concurrent experiments -both Figure 5A and 6C datasets were generated during the same experimental batch, though logically separated into two figures to enhance manuscript readability. Meanwhile, the core conclusions remain fully valid, as all results were independently validated with three biological replicates and the loading control duplication represents a presentational oversight rather than compromised data integrity. The corrected Figure 6C and its caption "Western blot analysis for the expression of stemnessassociated genes in MDA-MB-231 and CAL-51 cells after DTL knockdown" appear below.The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Keywords: DTL, Glycolysis, L-lactate, breast cancer, progression

Received: 16 May 2025; Accepted: 10 Jun 2025.

Copyright: © 2025 Lv, Liu, Li and Cao. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

* Correspondence:
Mengzhu Lv, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Laboratory of Molecular Oncology, Peking University Cancer Hospital and Institute, Beijing, China, Beijing, China
Yiren Cao, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Laboratory of Molecular Oncology, Peking University Cancer Hospital and Institute, Beijing, China, Beijing, China

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.