CORRECTION article

Front. Pharmacol., 13 September 2019

Sec. Neuropharmacology

Volume 10 - 2019 | https://doi.org/10.3389/fphar.2019.01037

Corrigendum: Scopoletin Suppresses Activation of Dendritic Cells and Pathogenesis of Experimental Autoimmune Encephalomyelitis by Inhibiting NF-κB Signaling

  • National Engineering Laboratory for Resource Development of Endangered Crude Drugs in Northwest China, The Key Laboratory of Medicinal Resources and Natural Pharmaceutical Chemistry, The Ministry of Education, College of Life Sciences, Shaanxi Normal University, Xi’an, China

In the published article, author Zhe-Zhi Wang’s identity was incorrect. Instead of “co-author,” it should be “co-corresponding author.” The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Summary

Keywords

scopoletin, experimental autoimmune encephalomyelitis, multiple sclerosis, dendritic cells, NF-κB signaling

Citation

Zhang F, Zhang Y, Yang T, Ye Z-Q, Tian J, Fang H-R, Han J-J, Wang Z-Z and Li X (2019) Corrigendum: Scopoletin Suppresses Activation of Dendritic Cells and Pathogenesis of Experimental Autoimmune Encephalomyelitis by Inhibiting NF-κB Signaling. Front. Pharmacol. 10:1037. doi: 10.3389/fphar.2019.01037

Received

13 August 2019

Accepted

14 August 2019

Published

13 September 2019

Approved by

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Volume

10 - 2019

Updates

Copyright

*Correspondence: Zhe Zhi Wang, ; Xing Li,

†These authors have contributed equally to this work

This article was submitted to Neuropharmacology, a section of the journal Frontiers in Pharmacology

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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