CORRECTION article

Front. Oncol., 10 January 2025

Sec. Gynecological Oncology

Volume 14 - 2024 | https://doi.org/10.3389/fonc.2024.1545180

Corrigendum: DYNLT3 overexpression induces apoptosis and inhibits cell growth and migration via inhibition of the Wnt pathway and EMT in cervical cancer

  • 1. Center of Uterine Cancer Diagnosis and Therapy Research of Zhejiang Province, Department of Obstetrics and Gynecology, The Second Affiliated Hospital of Wenzhou Medical University, Wenzhou, China

  • 2. Department of Obstetrics and Gynecology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China

In the published article, a minor error was identified in Figure 5C. Due to carelessness during the preparation of the figures, the panels labeled “Invasion, SiHa, sh2-Dynlt3” and “Migration, CaSki, sh1-Dynlt3” were incorrectly pasted. The corrected Figure 5 and its caption appear below.

Figure 5

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Statements

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

DYNLT3, cervical cancer, proliferation, apoptosis, invasion, migration

Citation

Zhang J, Shen Q, Xia L, Zhu X and Zhu X (2025) Corrigendum: DYNLT3 overexpression induces apoptosis and inhibits cell growth and migration via inhibition of the Wnt pathway and EMT in cervical cancer. Front. Oncol. 14:1545180. doi: 10.3389/fonc.2024.1545180

Received

14 December 2024

Accepted

17 December 2024

Published

10 January 2025

Volume

14 - 2024

Edited and reviewed by

Joon-Yong Chung, National Cancer Institute (NIH), United States

Updates

Copyright

*Correspondence: Xueqiong Zhu, ; Xuejie Zhu,

†These authors have contributed equally to this work

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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