CORRECTION article
Front. Oncol.
Sec. Head and Neck Cancer
Volume 15 - 2025 | doi: 10.3389/fonc.2025.1651340
Correction: Heterotypic neutrophil-in-tumor structure: a novel pathological feature first discovered in the tissues of OPSCC
Provisionally accepted
- 1Henan Cancer Hospital Affiliated Cancer Hospital of Zhengzhou University, Zhengzhou, China
- 2The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China
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Corrigendum on: Fan J, et al. Heterotypic neutrophil-in-tumor structure: a novel pathological feature first discovered in the tissues of OPSCC. Front Oncol. 2022 Aug 16; 541878. DOI: 10.3389/fonc.2022.807597. Error in FigureIn the published article, there were errors in Figure 5B as published. In original Figure 5, the fluorescent staining images of Figure 5B was vague, and the third image from the left in the first row of Figure 5B was mistakenly used in the work of another colleague in my lab, so I decided to delete Figure 5B. The updated Figure 5 appear below.Error in Figure LegendIn the published article, there was an error in the legend for Figure 5 as published. Because original Figure 5B was decided to be deleted, the relevant legend for original Figure 5B was to be deleted accordingly. The original legend numbers (C) and (D) for Figure 5C and 5D were changed to (B) and (C), respectively. The corrected legend appears below. Legend for Figure 5. Fluorescent staining result of typical hNiT structures formed between SCC-15 and HL-60 cells which negatively correlate with CDKN2A expression. (A) Fluorescent staining images of typical hNiT structures formed between SCC-15-H or SCC-15-L and HL-60 cells(SCC-15-H: subpopulation of SCC-15 with the high ability to internalize more HL-60 cells to form hNiT; SCC-15-L: subpopulation of SCC-15 with the low ability to internalize less HL-60 cells to form hNiT). Scale bars: 10μm. (B) The hNiT formation in H1, H2, L1 and L2 subpopulations of SCC-15 cells. (C) The CDKN2A expression in H1, H2, L1 and L2 subpopulations of SCC-15 cells (H1 and H2: subpopulations of SCC-15 with the high ability to internalize more HL-60 cells to form hNiT; L1 and L2: subpopulations of SCC-15 with the low ability to internalize less HL-60 cells to form hNiT).The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Keywords: heterotypic neutrophil-in-tumor structure, Oropharyngeal squamous cell carcinoma, p16, novel pathological feature, disease-specific survival
Received: 21 Jun 2025; Accepted: 30 Jun 2025.
Copyright: © 2025 Fan, Li, Fang, Yang, Zhang, Du, Shanting and Luo. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
* Correspondence: Jie Fan, Henan Cancer Hospital Affiliated Cancer Hospital of Zhengzhou University, Zhengzhou, China
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