CORRECTION article

Front. Pharmacol., 18 January 2023

Sec. Inflammation Pharmacology

Volume 13 - 2022 | https://doi.org/10.3389/fphar.2022.1108989

Addendum: A novel N-Arylpyridone compound alleviates the inflammatory and fibrotic reaction of silicosis by inhibiting the ASK1-p38 pathway and regulating macrophage polarization

  • 1. Department of Respiratory Medicine, The Second Hospital of Jilin University, Jilin, China

  • 2. National Center for Respiratory Medicine, National Clinical Research Center for Respiratory Diseases, Department of Pulmonary and Critical Care Medicine Center of Respiratory Medicine, Chinese Academy of Medical Sciences, Peking Union Medical College, China-Japan Friendship Hospital, Capital Medical University, Institute of Respiratory Medicine, Beijing, China

  • 3. Department of Pulmonary and Critical Care Medicine, China-Japan Friendship School of Clinical Medicine, Peking University, Beijing, China

  • 4. Institute of Clinical Medical Sciences, China-Japan Friendship Hospital, Beijing, China

  • 5. State Key Laboratory of Medical Molecular Biology, School of Basic Medicine Peking Union Medical College, Institute of Basic Medical Sciences Chinese Academy of Medical Sciences, Beijing, China

Missing information

Add information about chemical formula, Table 2, polarization and antibody list.

TABLE 2

AntibodiesCat No.ManufacturerSources of species
Fibronectinab2413abcamRabbit
Collagen-Ⅰ66761-1-IgproteintechMouse
IκBα#4814cell signaling technologyMouse
p38#8690cell signaling technologyRabbit
P-p38#4511cell signaling technologyRabbit
Arginase-1ab233548abcamRabbit
ASK-167072-1-IgproteintechRabbit
P-ASK-128846-1-APproteintechRabbit
NF-kBab32536abcamRabbit
P-NF-kBab76302abcamRabbit
iNOS18985-1-APproteintechRabbit
β-Actinab8226abcamMouse

(Antibody list) Primary antibodies for Western Blot.

Chemical formula

Chemical structure of AKEX0011

Molecular formula C18H14N5O2F3, mass 389.3 g/mol, CAS:1590403-33-0.

Polarization

RAW 264.7 cells are a macrophage-like, Abelson leukemia virus-transformed cell line derived from BALB/c mice. This macrophage cell line is commonly used to study phagocytosis, apoptosis, inflammation, as well as M1/M2 polarization. Polarization of M1/M2 phenotype can be determined by the expression of specific M1 (CD80, CD86) and M2 (CD206, CD163) markers detected by flow cytometry. In the in vitro experiments of our study, we detected the expression of CD86 and CD163 in RAW264.7 cells of each group by flow cytometry to explore macrophage polarization. Our flow cytometry results confirmed that silica induced polarization of RAW264.7 from M0 macrophages toward certain M1 subtype (F4/80 + CD86+), but have no effect on the polarization towards M2 subtype (F4/80 + CD163+).

Statements

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

AKEX0011, macrophage polarization, pirfenidone, pulmonary fibrosis, silicosis

Citation

Fan M, Xiao H, Song D, Zhu L, Zhang J, Zhang X, Wang J, Dai H and Wang C (2023) Addendum: A novel N-Arylpyridone compound alleviates the inflammatory and fibrotic reaction of silicosis by inhibiting the ASK1-p38 pathway and regulating macrophage polarization. Front. Pharmacol. 13:1108989. doi: 10.3389/fphar.2022.1108989

Received

27 November 2022

Accepted

29 November 2022

Published

18 January 2023

Volume

13 - 2022

Edited and reviewed by

Paola Patrignani, University of Studies G. d'Annunzio Chieti and Pescara, Italy

Updates

Copyright

*Correspondence: Jing Wang, ; Huaping Dai, ; Chen Wang,

This article was submitted to Inflammation Pharmacology, a section of the journal Frontiers in Pharmacology

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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