CORRECTION article
Front. Pharmacol.
Sec. Renal Pharmacology
Volume 16 - 2025 | doi: 10.3389/fphar.2025.1710116
Correction: Astragaloside IV Alleviates Tacrolimus-Induced Chronic Nephrotoxicity via p62-Keap1-Nrf2 Pathway
Provisionally accepted- 1Wuhan Children's Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
- 2Huazhong University of Science and Technology Tongji Medical College Union Hospital, Wuhan, China
- 3Wuhan Fourth Hospital, Wuhan, China
- 4Huazhong University of Science and Technology Tongji Medical College Tongji Hospital, Wuhan, China
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There were mistakes in figure 3D and figure 6E as published. In figure 3D, the image of Tac group (the second group) was misused. In figure 6E, the western blot of p62 was misused. The corrected figure 3D and figure 6E appear below. Last Name author1 et al. Frontiers Media SA 2 FIGURE 3. The cell protective and ROS scavenge effect of Astragaloside IV in HK-2 cells. (D) The levels of intracellular ROS were detected with 2', 7'-dichlorodihydrofluorescein diacetate (H2DCFDA) assay. FIGURE 6. Astragaloside IV increased Keap1 degradation and p62 phosphorylation in vivo. (E) The p62 as well as phosphorylated p62 protein levels.
Keywords: Astragaloside IV, Tacrolimus, Chronic nephrotoxicity, p62-Keap1-Nrf2 pathway, Oxidative Stress
Received: 21 Sep 2025; Accepted: 02 Oct 2025.
Copyright: © 2025 Gao, Du, Liu, Xu, Liu, Guan and Zhang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
* Correspondence: Chengliang Zhang, clzhang@tjh.tjmu.edu.cn
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