In the published article, there was an error in Figure 6 as published. In our four staining methods, the magnification is inconsistent, and the scale annotation is not consistent with the original image. Therefore, we uniformly choose 50μm Display at magnification. The corrected Figure 6 and its caption appear below.
Figure 6
H&E staining indicated obvious fibrosis lesion in FIB mice and apparent tissue inflammation, which were further aggravated in DNTs, along with severe liver injury, while MCC950 improved liver fibrosis degree. Masson and sirius-red staining results indicated that, DNTs promoted liver fibrosis in mice, the expression of collagen fibers and collagens further increased in hepatic tissues, higher than that in FIB, while MCC950 suppressed such changes. Moreover, IHC also revealed that DNTs promoted α-SMA expression. *P<0.05 compared with Control group, #P<0.05 compared with FIB group.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Statements
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Summary
Keywords
double-negative T cells, liver fibrosis, NLRP3, TNFR1, hepatic stellate cells
Citation
Yang Y, Sheng Y, Wang J, Zhou X, Li W, Zhang C, Guo L and Han C (2023) Corrigendum: Double-negative T cells regulate hepatic stellate cell activation to promote liver fibrosis progression via NLRP3. Front. Immunol. 14:1340576. doi: 10.3389/fimmu.2023.1340576
Received
18 November 2023
Accepted
23 November 2023
Published
01 December 2023
Volume
14 - 2023
Edited and reviewed by
Hai-Jing Zhong, Jinan University, China
Updates
Copyright
© 2023 Yang, Sheng, Wang, Zhou, Li, Zhang, Guo and Han.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Chenyang Han, 691513770@qq.com
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.