In the original article, there was a mistake in Figure 1 as published. Nociceptors (C-fibers) and Proprioceptors (Aδ-fibers) instead of Nociceptors (Aδ/C fibers) and Proprioceptors (Aα fibers). The corrected Figure 1 appears below. The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Figure 1
Cellular elements involved in pain transmission from the peripheral to the central nervous system (CNS). (Box 1) The pain (thermal, high pressure, mechanical, chemical) information is first detected by the receptors located at the level of free nerve endings of primary sensory neuron (PSN) fibers. (Box 2) Then, it is conveyed by the dendrites of these neurons, components of dorsal root ganglia (DRG), to the dorsal horn of spinal cord where it is transmitted to the dendrites of secondary sensory neurons (SSNs). (Box 3) Finally, it is brought to the hypothalamus via the tertiary sensory neurons (TSNs) whose cell bodies constitute, in part, the brain cortex.
Statements
Conflict of interest
TG and MP are current or former employees of Sanofi. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Summary
Keywords
voltage-gated sodium channels, NaV1.7 channel subtype, spider toxins, pain, dorsal root ganglia neurons, electrophysiology
Citation
Gonçalves TC, Benoit E, Partiseti M and Servent D (2018) Corrigendum: The NaV1.7 Channel Subtype as an Antinociceptive Target for Spider Toxins in Adult Dorsal Root Ganglia Neurons. Front. Pharmacol. 9:1241. doi: 10.3389/fphar.2018.01241
Received
05 October 2018
Accepted
12 October 2018
Published
24 October 2018
Volume
9 - 2018
Edited and reviewed by
Yuri N. Utkin, Institute of Bioorganic Chemistry (RAS), Russia
Updates
Copyright
© 2018 Gonçalves, Benoit, Partiseti and Servent.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Denis Servent denis.servent@cea.fr
This article was submitted to Pharmacology of Ion Channels and Channelopathies, a section of the journal Frontiers in Pharmacology
Disclaimer
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