Abstract
Traditional public opinion diffusion models generally assume interactions between individuals as binary pair-wise effects, which struggle to capture the higher-order complexities of multi-group interactions in social networks—such as group discussions in WeChat and topic reposting on Weibo. Moreover, these models fail to adequately depict the nonlinear trust accumulation mechanisms and individual heterogeneity inherent in the diffusion process. Therefore, the paper proposes a hypergraph-based Hyper-S2IR model for disseminating public opinion. The “Goebbels effect” is operationalized by leveraging the hypergraph structure: a susceptible node’s risk of infection is proportional to its hyperdegree, mathematically representing the cumulative exposure to information from multiple sources within different hyperedges. Our model introduces two types of communicators (HI1 and HI2) with different motivations and capabilities, thereby systematically depicting the inherent heterogeneity of the communication group. Through theoretical derivation, we derive a novel basic reproduction number R0 that explicitly incorporates the hyperdegree distribution of the hypergraph. This R0 provides a threshold for dissemination dynamics: When R0 > 1, the public opinion will continue to spread and converge to a stable public opinion prevalence equilibrium point; when R0 < 1, the public opinion will gradually disappear. Critically, the expression for R0 reveals how higher-order group interactions, encoded in the hyperdegree, fundamentally alter the spreading threshold compared to traditional pairwise networks. Numerical simulations verify the theoretical conclusions and demonstrate that the hypergraph structure significantly accelerates the spread and expands the scale of public opinion compared to traditional network structures. This work provides theoretical support and a quantitative basis for analyzing public opinion dissemination mechanisms and formulating intervention strategies.
1 Introduction
With the increasing penetration and rapid development of the Internet, social networking platforms such as Weibo and WeChat have become primary channels for public communication. Information spreads on these platforms at unprecedented speed, and both the diversity and volume of online content have expanded by orders of magnitude compared with earlier media environments. In this context, public opinion often emerges from the combined effects of social environments, individual psychology, and information transmission. Accordingly, understanding, monitoring, guiding, and responding to public opinion has become a major challenge for individuals, institutions, and governments. Research on public opinion dissemination, therefore, carries substantial theoretical significance and practical value: it not only enhances the ability to interpret communication phenomena in the new-media era but also provides scientific decision support and actionable guidance for social governance, organizational management, and national development [1].
At its core, public opinion dissemination is a process of information transmission, whose propagation mechanism shares notable similarities with infectious disease spread. Consequently, early studies of public opinion diffusion often drew on classical epidemic models. The compartmental modeling framework was first proposed by Kermack and McKendrick [2], who studied outbreaks such as the plague and assumed homogeneous mixing, instantaneous contact, and no dependence on interaction history. Building on this framework, a series of classic epidemic models have been developed, including SI, SIS, SIR, SIRS, and SEIR models, among others [3–5].
A seminal model for public opinion dissemination is the Daley–Kendall model proposed in the 1960s [6], whose basic mechanism parallels the SIR model; Maki and Thomson further refined it in 1973 [7]. With growing recognition that network structure shapes diffusion dynamics, researchers subsequently proposed public opinion dissemination models based on complex networks. In 2001, Pastor-Satorras and Vespignani analyzed spreading processes on regular, random, and small-world networks and provided theoretical evidence for the existence of non-zero thresholds [8]. Meanwhile, Zanette [9, 10] was among the first to investigate public opinion spreading on complex networks, arguing that a threshold θ exists in small-world networks: when spreading parameters fall below θ, public opinion will eventually die out. These studies collectively underscore that network topology can significantly influence dissemination dynamics.
Despite these advances, most existing models still rely on a pairwise-interaction paradigm, assuming that individual interactions occur only between two nodes. In real online environments, however, interactions frequently take place at arbitrary scales, ranging from one-to-one communication to information diffusion within groups of varying sizes—for example, discussions in WeChat group chats or the collective reposting and commenting around Weibo topics. In addition, public opinion diffusion is often accompanied by the so-called “Goebbels effect,” whereby individuals may initially doubt a message but gradually increase their trust after repeated exposure from different sources, eventually accepting it as true [11, 12]. Repeated exposure and group interactions are thus key drivers of state transitions in public opinion dynamics. Nevertheless, prevailing models exhibit clear limitations in representing these mechanisms. First, by remaining grounded in pairwise interactions, they struggle to describe higher-order and synchronous diffusion within groups. Second, they often assume fixed transition probabilities, thereby oversimplifying the nonlinear process through which trust is dynamically reinforced via repeated exposure, as conceptualized by the Goebbels effect.
The Goebbels effect reveals a fundamentally nonlinear process of trust formation at both individual and group levels. Cognitively, it can be interpreted as a dynamic evidence-accumulation mechanism, in which repeated exposure nonlinearly shifts beliefs by reducing cognitive inertia and status quo bias in decision-making [13]. From a modeling perspective, it also suggests a “major–minor factor” structure in trust formation: exposure frequency serves as the primary driver, while individual sensitivity modulates its impact [14]. From a social-dynamics viewpoint, the effect aligns with the logic that moral alignment fosters group consensus, emphasizing how repeated micro-level interactions can lead to macro-level consensus formation [15]. Therefore, modeling the Goebbels effect is not merely a refinement beyond simple probabilistic transitions, but a more faithful representation of the cognitive and social mechanisms underlying public opinion formation [16].
Furthermore, many models treat spreaders as homogeneous, overlooking heterogeneity in motivation, capability, and social influence. Consequently, building an integrated framework that jointly captures group interactions, nonlinear trust accumulation, and spreader heterogeneity has become a central challenge in modeling public opinion diffusion in real-world networks [17]. The homogeneity assumption is an oversimplification that weakens model realism and predictive validity: in practice—especially for contentious topics—spreaders differ markedly in emotional tone, motivation, and persuasive power. Emotionally aroused spreaders may repost rapidly and reflexively, whereas deliberative spreaders are more likely to fact-check and engage in reasoned discussion. Such heterogeneity fundamentally alters macroscopic diffusion outcomes, affecting diffusion speed, scale, and persistence, and can yield phenomena such as multi-peaked diffusion curves or the sustained coexistence of competing narratives—patterns that homogeneous-mixing models cannot capture. Hence, moving beyond homogeneity to explicitly model communicator heterogeneity is essential for developing more realistic and predictive diffusion models.
Hypergraphs [18], as an important extension of graph theory for modeling higher-order interactions, allow a single hyperedge to connect multiple nodes. This property makes hypergraphs well-suited for representing group interaction units such as WeChat groups and topic communities, thereby enabling a more accurate description of synchronous information diffusion within groups [19]. Motivated by this, and to better reflect intrinsic differences among communicators, this paper draws on the S2IR model [20] and explicitly distinguishes a second type of disseminator (I2). A new hypergraph-based public opinion dissemination model, termed Hyper-S2IR, is proposed, and its dynamical behavior is examined through theoretical analysis and simulation-based validation.
The main contributions of this article are as follows:
Hyper-S2IR Model Construction: Based on hypergraph theory, the Hyper-S2IR public opinion dissemination model is constructed to describe the multi-dimensional relationships among nodes. A second type of disseminator (I2) is added to distinguish individuals with different dissemination motivations and capabilities. By parameterizing and quantifying the dynamic transformation mechanism of the two types of disseminators, the practical interpretability of the model is enhanced.
Theoretical Analysis of Dynamic Behavior: By deriving the expression for the basic reproduction number R0, establish the threshold criteria for public opinion dissemination. Through theoretical analysis, we rigorously prove the existence of both the no-public opinion equilibrium point and the public opinion equilibrium point. Based on the analysis of the eigenvalues of the Jacobian matrix, the local asymptotic stability conditions are clearly defined. Further, by using the Lyapunov function and Lasalle’s invariance principle, the global asymptotic stability of the no-public opinion equilibrium point is verified.
Multi-dimensional verification through numerical simulation: Numerical experiments were conducted on multiple sets to verify the theoretical results. The system compared the dissemination dynamics behaviors under the hypergraph structure and the traditional network structure. The simulation results showed that the hypergraph structure could significantly accelerate the spread of public opinions, expand the scale of the spread, reveal the influence of the network structure on the dissemination dynamics, and the sensitivity of key parameters to the dissemination process. This can provide a quantitative basis for public opinion intervention strategies.
2 Related work
2.1 Hypergraph and hypergraph applications
In hypergraphs, a hyperedge can simultaneously connect an arbitrary number of nodes, enabling a natural representation of multi-member group structures such as WeChat groups, online communities, and collaborative teams. Owing to this expressive power, hypergraphs have been widely applied in social network analysis, recommender systems, and knowledge graphs [21]. In dissemination dynamics, hypergraphs provide a principled framework for describing real-world group diffusion, where information often spreads synchronously within groups rather than solely through pairwise contacts. In recent years, classical spreading models have been successfully extended to hypergraph settings [22–24]. However, although these studies highlight the advantages of hypergraphs for modeling population-level transmission, most of them retain the classical SIS/SIR-style compartmental architecture and thus largely overlook intrinsic heterogeneity among transmitters.
Recent progress in graph learning further supports the value of hypergraph-based modeling. For tasks that require complex relational reasoning, such as fact verification, approaches that strengthen structural connectivity have been shown to yield superior performance [25]. Similarly, models that explicitly capture higher-order dependencies often achieve improved accuracy and robustness, particularly in low-label regimes [26]. These findings jointly suggest that incorporating higher-order relational structures is not only theoretically well motivated but also empirically beneficial for modeling and inference in complex information environments.
Figure 1 illustrates an example of a hypergraph and hypergraph dissemination.
FIGURE 1
In Figure 1A, the node set V = {v1, v2, … , v9} and the hyperedge set E = {E1, E2, E3, E4}. E1 = {v1, v2, v9}, E2 = {v2, v3, v4, v5, v6}, E3 = {v6, v7}, and E4 = {v7, v8, v9}. Consequently, the hyperdegree of v1, v3, v4, v5, and v8 is 1, while that of v2, v6, v7 and v9 is 2. Figure 1B illustrates an example of hypergraph dissemination, where red nodes represent infected nodes and blue nodes denote susceptible nodes. The probability that the infected node v6 infects the susceptible node v7 through a hyperedge of size 2 is β2. If a hyperedge E4 of size 3 contains two infected nodes v8 and v9, then they will infect the unique susceptible node through this hyperedge, with a probability of β3.
2.2 Fundamental theory of dissemination models
With continued advances in understanding information dissemination mechanisms, communication models have evolved from early homogeneous and deterministic frameworks toward more sophisticated systems that can represent the heterogeneity and dynamism of complex social environments. Recent studies have made substantial progress in developing integrated analytical frameworks that incorporate network heterogeneity and behavioral complexity. Nevertheless, several issues remain open. First, although complex-network and multilayer-network approaches can effectively characterize binary relationships between nodes [27, 28], most models are still fundamentally grounded in pairwise interactions and thus have difficulty capturing the synchronous group interactions that frequently occur in real-world settings. Second, while psycho-behavioral coupling mechanisms have been introduced to partially account for individual differences, many traditional models still treat communicators as a largely homogeneous population, providing insufficient characterization of within-group differentiation in motivation, emotional tendencies, and behavioral patterns [29, 30].
In real-world public opinion dissemination—especially during sudden public incidents, social issue debates, or political elections—the information environment is rarely singular. Instead, it is typically composed of multiple competing, opposing, or coexisting viewpoints, narratives, and emotional inclinations. The S2IR model proposed by Wang et al. [31] offers a useful reference for representing such heterogeneity by distinguishing two categories of disseminators.
The S2IR model divides the population into the following four categories:
Susceptible (S): Individuals who have not yet encountered or taken notice of any dominant narrative;
Infected-A (IA): Individuals who endorse and actively disseminate Narrative A;
Infected-B (IB): Individuals who endorse and actively disseminate Narrative B;
Recovered (R): Individuals who are aware of relevant information but have ceased dissemination.
The dissemination rules for the S2IR model are as follows:
Individuals within the population are uniformly mixed, with each having an equal opportunity to interact with others;
When susceptible individuals come into contact with the disseminators of Narrative A, they will acquire and believe this narrative at a dissemination rate of β, thereby transforming into the same type of disseminators.
When different disseminators come into contact, there is a possibility of direct mutual persuasion and change of stance. An individual disseminating narrative B can convince an individual disseminating narrative A with a conversion rate of σAB, causing the former to abandon their original viewpoint and instead believe and disseminate narrative B. An individual disseminating narrative A can convince an individual disseminating narrative B with a conversion rate of σBA, causing the latter to abandon their original viewpoint and instead believe and disseminate narrative A.
After spreading for a period of time, disseminators may stop spreading due to loss of interest, realization of false information, or forgetting, with an immunity rate γ. Immune individuals will no longer believe or spread.
The state transition diagram for the S2IR model is shown in Figure 2.
FIGURE 2
The differential equation form of the S2IR model is shown in Equation 1.
In the equation, S(t), IA(t), IB(t), and R(t) denote the densities of the four population categories at time t, satisfying S(t)+ IA(t)+ IB(t)+ R(t) = 1. β represents the transmission rate, γ denotes the immunity rate, while σAB and σBA represent the conversion rates between transmission and immunity states.
Mathematical formalization of the Goebbels effect. To mechanistically capture the “Goebbels effect”—where repeated exposure induces cumulative trust—a general formalization is introduced. The key premise is that the total infection pressure experienced by a susceptible individual i, denoted by Λi(t), is a monotonically increasing function of the effective exposure count Ni(t). The specific formula is shown in Equation 2.
Here, Ni(t) represents the effective and independent number of information exposures that individual i has experienced by time t. The function f (·) is a monotone increasing trust-accumulation function that maps exposure counts to a trust level, f (0) = 0. The term ρ(t) denotes the baseline infection pressure per exposure, reflecting factors such as message virality or contextual intensity.
To situate the present study within the literature and clarify its contributions, Table 1 summarizes representative opinion dissemination models along three dimensions: underlying network structure, treatment of spreader types, and primary analytical focus. As shown, prior work has made notable progress in modeling heterogeneous spreaders on graphs [17, 28, 31] and homogeneous spreaders on hypergraphs [22, 32]. However, a critical gap remains in frameworks that can simultaneously capture (i) higher-order group interactions naturally represented by hypergraphs and (ii) intrinsic heterogeneity among communicators. The Hyper-S2IR model is developed to address this gap by integrating these two essential aspects, thereby providing a more nuanced and realistic characterization of public opinion dissemination.
TABLE 1
| Topics | Structure | Spreader types | Analysis |
|---|---|---|---|
| SIR [10] | Graph | Homogeneous | Stability |
| S2IR [31] | Graph | Heterogeneous | Stability |
| SIR variant [17] | Graph | Homogeneous | Stability |
| SIS with time delay [33] | Graph | Homogeneous | Stability |
| SLCIR [28] | Graph | Heterogeneous | Stability |
| Hyper-SIR [32] | Hypergraph | Homogeneous | Stability and dynamics |
| Hyper-SIS [22] | Hypergraph | Homogeneous | Stability and dynamics |
| Hyper-S2IR | Hypergraph | Heterogeneous | Stability and dynamics |
Comparative summary of dissemination model features from the literature.
3 Hyper-S2IR model
3.1 Hyperedge-degree dissemination
In the hypergraph, the hyperdegree of node vi represents the breadth of its participation in group interactions, defined as the set of all hyperedges to which the node belongs. The hyperdegree vector of node vi is represented as an (m-1)-dimensional vector, as shown in Equation 3.
Among them, ki(m) represents the mth degree of the node. It represents the number of hyperedges of size m for the node, where m = 2, 3, …, M. The hyperdegree is arranged in ascending order of each degree. For example, [5, 2, 0, …, 0] is placed before [6, 2, 0, …, 0], [2, 3, 1, …, 0] is placed before [2, 3, 2, …, 0], and [5, 1, 2, 0, 1, …, 0] is placed after [5, 1, 0, 2, 1, …, 0] [32].
In Figure 1A, the excess degrees of v1 and v8 are [0,1,0,0], the excess degree of v2 is [0,1,0,1], the excess degrees of v3, v4, and v5 are [0,0,0,1], and the excess degrees of v6, v7, and v9 are [1,0,0,1], [1,1,0,0], and [0,2,0,0] respectively. According to the above sorting method and arranging in ascending order of excess degree numbers, then K1 = [0,0,0,1], In addition, K2 = [0,1,0,0], K3 = [0,1,0,1], K4 = [0,2,0,0], K5 = [1,0,0,1], and K6 = [1,1,0,0].
3.2 Model assumptions
To describe dissemination issues in the Hyper-S2IR model, the following node states are defined as follows:
Hyper-Susceptible (HS): A node that is not yet aware of the public opinion, possibly being influenced by multiple dissemination nodes simultaneously through the associated hyperedge, and thus coming into contact with the public opinion. The HS node can simultaneously belong to multiple hyperedges containing dissemination nodes, thereby facing dissemination pressure from multiple directions.
Hyper-Infected1 (HI1): Nodes that have become aware of public opinion and propagate the first category of emotion within hyperedges. Their dissemination behaviors can simultaneously influence multiple susceptible nodes within the same hyperedge through a single hyperedge, enabling one-to-many emotional diffusion.
Hyper-Infected2 (HI2): Nodes that have detected public opinion and propagate the second category of emotion within hyperedges. Similar to HI1, HI2 can simultaneously transmit emotion to multiple adjacent nodes within its hyperedge. Both types of propagating nodes may coexist within the same hyperedge, competing to influence susceptible nodes.
Hyper-Recovered (HR): Nodes that are aware of the public opinion but have temporarily withdrawn from dissemination, possessing a degree of short-term immunity. Within hypergraphs, HR nodes may still reside within hyperedges, yet currently do not participate in opinion dissemination. Consequently, they are less susceptible to being re-influenced by other propagating nodes within the same hyperedge.
Define the dissemination rules as follows:
Individuals within each hyperedge are uniformly mixed, with each individual having an equal opportunity to interact with others within the same hyperedge;
Susceptible node HS, upon receiving information from infected node HI1 (or HI2), may believe it and propagate the information with probability α;
Infected nodes HI1 and HI2, upon receiving information from the other, may believe it and propagate opposing information with probabilities β1 and β2. They may lose interest in transmitting information or realize the information is erroneous, after which they become immune nodes HR with probability γ;
Over time, immune nodes HR forget the information and become susceptible nodes HS with probability ω.
The meanings of the various parameters involved in the Hyper-S2IR dissemination model are shown in Table 2.
TABLE 2
| Parameter | Explanation |
|---|---|
| α | The transition probability from HSKi(t) to HInKi(t) |
| β1 | The transition probability from HI1Ki(t) to HI2Ki(t) |
| β2 | The transition probability from HI2Ki(t) to HI1Ki(t) |
| γ | Recovery rate |
| ω | Forgetfulness rate |
| αm | The infection rate of hyperedges of size m |
| P(Ki) | The distribution of hyperdegree Ki |
| Average hyperdegree |
Parameter explanation.
The state transition diagram of the Hyper-S2IR dissemination model is shown in Figure 3.
FIGURE 3
Hyper-S2IR model dynamic equations are as follows:
Here, HSKi(t), HI1Ki(t), HI2Ki(t), and HRKi(t) denote the densities of the four types of state nodes with hyperdegree Ki at time t, respectively. α, β1, β2, γ, and ω are non-negative constant parameters.
The system of dynamic Equation 4 describes the rate of change for each node state. The physical interpretation of each equation is as follows:
dHSKi(t)/dt: The population decreases as nodes are infected by contact with HI1 or HI2 nodes in hyperedges (-||Ki||(Θ1+Θ2)HS), and increases as recovered nodes lose immunity (+ωHR).
dHI1Ki(t)/dt: The population grows with new infections of susceptible nodes by HI1 nodes (+α||Ki||HSΘ1). It decreases through spontaneous recovery (-γHI1) and state transitions to HI2 due to competition (-β1HI1Θ2), but can gain nodes converted from HI2(+β2HI2Θ1).
dHI2Ki(t)/dt: This equation is symmetric to the HI1 equation, describing the analogous dynamics for the second type of infected nodes.
dHR(t)/dt: The population increases from susceptible nodes that do not become spreaders after contact (+(1-α)||Ki||(Θ1+Θ2)HS) and from the recovery of both infected types (+γ(HI1+HI2)). It decreases as nodes lose immunity and re-enter the susceptible compartment (-ωHR).
The infection probability function Θn(t) denotes the probability that any given hyperedge points to an infected node, as shown in Equation 5.
Here, denotes the intensity of public opinion dissemination by infected nodes of degree Ki. Let M be the size of the largest hyperedge in the hypergraph, as shown in Equation 6.
Here, αm denotes the infection rate of hyperedges of size m, signifies the probability at the current time step that an infected neighbor node of degree Ki is connected to a given node. .
P(Ki|Kj) denotes the conditional probability that a node with degree Ki is connected to a node with degree Kj, as shown in Equation 7.
Here, is the average hyperdegree. Therefore,
Obviously, , then
Here, P(Ki) is the hyperdegree distribution.
Remark 1The Hyper-S2IR model is a generalization that incorporates higher-order interactions. Notably, the classical S2IR model on graphs [31] is a specific instance of our model. This is achieved when the maximum hyperedge size is set to M = 2, effectively reducing the hypergraph to a standard graph where all interactions are pairwise. In this scenario:The hyperdegree of a node becomes equivalent to its standard degree in a network.The infection probability Θn(t) (Equation 5) simplifies, as the summation over hyperedge sizes m collapses to a single term for m = 2.Consequently, the dynamic Equation 4 reduce precisely to the form of the S2IR model. This establishes that our model seamlessly generalizes the pairwise interaction paradigm to account for group-based diffusion.
4 Model equilibrium point analysis
4.1 No-public opinion equilibrium point and proof of existence
In the Hyper-S2IR epidemic model, if all infection state variables are zero, then the no-public opinion equilibrium point E0 = {1,0,0,0}exists.
Proof. Substituting HI1Ki(t) = 0 and HI2Ki(t) = 0 into the model’s set of dynamic Equation 4 and setting the derivatives of all variables to zero, as shown in Equation 10.
From Equation 9, ϴ1* = ϴ2* = 0. Combining this with the constraint HSki*+ HI1ki*+HI2ki*+ HRki* = 1, as shown in Equation 11.
We obtain the result as shown in Equation 12.
For all groups i, the equilibrium point always exists and is independent of the parameter conditions; it is a self-evident solution of the model.
4.2 Derivation of the basic reproduction number R0
This paper employs a linearization analysis method for the infection subsystem to derive R0. Near the equilibrium point without public opinion, the dynamical equations governing the infection variables are linearized, and the corresponding Jacobian matrix M is constructed. By calculating the maximum eigenvalue of this matrix, the dominant growth rate of the system is obtained, thereby defining the basic reproduction number.
The specific derivation process of
R0is as follows:
Linearization of the Infection Subsystem Dynamical Equations.
Consider the system dynamics equations at the equilibrium points where HSKi = 1, HI1Ki = 0, HI2Ki = 0, and HRKi = 0. Express all variables as equilibrium values plus perturbation terms (e.g., HI1Ki = 0+δHI1Ki), retaining only first-order small terms. Since the proportion of susceptible individuals remains close to 1 near the no-public opinion equilibrium point, and its perturbation term is initially far smaller than the infection variable, HSKi can be approximated as the constant 1. Similarly, the cross-terms within the system of dynamical equations constitute higher-order infinitesimals when the infection variable is extremely small and may be neglected in preliminary linearized analyses.
The approximate equation for the dynamics of the infected subsystem is shown in Equation 13.
Substituting the self-consistent dissemination relationship
Equation 8into the dynamical equations yields a system of coupled linear differential equations for multiple variables, as shown in
Equation 14.
- 2.
Construct the Jacobian matrix.
Record all infection-related variables as a 2z-dimensional state vector as shown in Equation 15.
After linearization, the dynamic system Equation 13 is transformed as shown in Equation 16.
Here, M denotes the Jacobian matrix of the system, which is a 2z × 2z block matrix whose form is as shown in Equation 17.
Here, A and D represent the “self-consistent dissemination” and self-dissipation components of HI1Ki and HI2Ki, respectively, while B and C denote the linear coupling of cross-branches.
Block A: The self-consistent dissemination and loss of the HI1Ki on its own branch are described by the matrix elements as shown in Equation 18.
Block B and Block C: The cross-coupling between HI2Ki and HI1Ki, as revealed by the system of differential Equation 14, indicates no direct interaction coupling between them. The matrix values are thus expressed as in Equation 19.
Block D: The self-consistent dissemination and loss of the
HI2Kion its own branch are described by the matrix elements as shown in
Equation 20.
- 3.
Solving the eigenvalue equation.
Assuming the eigenvector is x = [x1, … ,xz]T and the eigenvalue is λ, we can obtain the result as shown in Equation 21.
Let , and simplifying, as shown in Equation 22.
Substituting xi into the derivation process of the definition of HS yields, as shown in Equation 23.
Eliminate
HSfrom both sides of the equation (where
HS≠ 0), we obtain:
- 4.
The final step in deriving R0
According to the threshold theorem for public opinion dissemination, when R0 = 1, the dissemination of public opinion reaches a critical point. At this juncture, the eigenvalues λ of the Jacobian matrix’s characteristic equation in the dissemination dynamics equation set are equal to zero. In summary, the definition formula for R0 is presented as shown in Equation 25.
When R0 > 1, the system possesses the capacity for sustained transmission; when R0 ≤ 1, the system ultimately returns to a state of no infection.
4.3 The public opinion equilibrium point and proof of existence
The equilibrium point of public opinion refers to the state in which, following prolonged system evolution, all state variables stabilize, and a proportionate number of infected individuals persist. This is characterized by the conditions HI1Ki > 0 and HI2Ki > 0 holding for all group i, reflecting the capacity of public opinion to sustain itself and endure over the long term within the network.
To calculate the epidemic equilibrium point of public opinion, set the derivatives of all variables with respect to time in the system of dynamic Equation 4 to zero, yielding the system of algebraic equations as shown in Equation 26.
Simplifying the system of Equation 26, we obtain:
The expressions for HI1Ki and HI2Ki in the simultaneous Equation 27 can be calculated as shown in Equation 28.
Combining HSki*+ HI1ki*+ HI2ki*+ HRki* = 1, calculate the equilibrium points of the system of equations. As shown in Equation 29.
Where ϴ1* and ϴ2* satisfy the self-consistency relationship as shown in Equation 30:
4.4 Theoretical analysis of the persistent circulation of public opinion
Let the state variable X be defined as shown in Equation 31.
The dynamic Equation 4 is converted as shown in Equation 32.
If the basic reproduction number R0 > 1, then the circulation of public opinion will inevitably converge towards a stable state of sustained epidemic circulation, meaning that a no-public opinion equilibrium point for sustained epidemic circulation exists.
Proof. The state space of the dissemination system (4) is defined as shown in Equation 33.
Set Ω denotes a compact, bounded, and closed convex set. When conducting linear perturbation analysis near the no-public opinion equilibrium point E0 = {1,0,0,0}, the initial growth rate of the perturbation variable (i.e., the maximum eigenvalue of the Jacobian matrix Equation 17) has previously been explicitly derived as shown in Equation 34.
According to Equation 34, when R0 > 1, the linearized system’s maximum growth rate λmax of the infection variable is positive, indicating that the no-public opinion equilibrium point is unstable under these conditions, with a tendency for the infection variable to grow spontaneously. Furthermore, the dominant growth rate of the system’s initial small disturbance is greater than zero, signifying that the system will depart from the no-public opinion equilibrium point and move towards the interior of the state space Ω.
Define the mapping as shown in Equation 35.
Here, τ denotes a sufficiently small time step. Since the function F(X) is continuous and Ω is a convex set, T(X) is also continuous. By the Brouwer fixed-point theorem, the continuous function T(X) must possess at least one fixed point X* on the compact, bounded, closed convex set Ω, satisfying the relationship as shown in Equation 36.
Therefore, there must exist a non-zero steady-state solution X*, representing the equilibrium point at which the public opinion persists.
If the basic reproduction number R0 ≤ 1, then public opinion dissemination will inevitably tend towards a state of equilibrium with no public opinion, and no non-zero state of sustained public opinion prevalence will exist.
Proof. The fundamental reproduction number and dominant growth rate of the infection variables, as derived earlier, are shown in Equations 25, 34. It follows that when R0 ≤ 1, λmax ≤ 0, meaning all infection variables exhibit non-positive growth rates. Any infection disturbance, however small, will decay exponentially over time, as demonstrated in Equation 37.
Therefore, the no-public opinion equilibrium point serves as the stable point for global attraction. Under all initial conditions, the system ultimately converges to the no-public opinion equilibrium point E0 = {1,0,0,0}, with no non-zero equilibrium points for sustained opinion prevalence existing.
5 Stability analysis
If R0 < 1, the no-public opinion equilibrium point E0 is locally asymptotically stable; if R0 > 1, E0 is unstable.
Proof. Simplify the system dynamics Equation 4 as shown in Equation 38.
The Jacobian matrix of the dynamical equation system Equation 38 at point E0 is shown in Equation 39.
Let , then each block matrix is specifically defined as shown in Equation 40.
Therefore, the characteristic equation of matrix Equation 40 is given by Equation 41.
From Equation 41, it follows that J (E0) possesses z negative eigenvalues λ = −ω and 2z − 2 negative eigenvalues λ = −γ, with . Consequently, when R0 < 1, all roots of Equation 38 are negative, rendering E0 locally asymptotically stable. When R0 > 1, E0 is unstable.
If R0 > 1 and ϴ1* and ϴ2* satisfy certain conditions, the Public opinion equilibrium point E* is locally asymptotically stable.
Proof. The Jacobian matrix of the system of Equation 38 at E* is given by Equation 42.
Let , then each block matrix is defined as shown in Equation 43.
According to the theory of matrix similarity transformations [33], let the invertible matrix P serve as the similarity transformation matrix, with P and P−1 defined as shown in Equation 44.
By a similar transformation of the matrix J (E*), it has G = PJ (E*)P−1, as shown in Equation 45
Here, the block matrices G11, G22, and G33 are defined as shown in Equation 46.
Since the block matrix G possesses an upper triangular structure, its characteristic equation is determined by the characteristic equations of these diagonal blocks. The eigenvalues of matrix G constitute the union of the diagonal eigenvalues, with the characteristic equation being as shown in Equation 47.
The characteristic equation of the block matrix G11 is given by Equation 48.
If all roots of the characteristic equation of matrix G11 are negative, then the following condition must be satisfied: , as shown in Equation 49.
The characteristic equation of the block matrix G22 is given by Equation 50.
Where,
If all roots of the characteristic equation of matrix G22 are negative, then the conditions and must be satisfied, as demonstrated in the derivation process shown in Equation 51.
From Equation 51, it can be seen that when , all eigenvalues are negative.
The characteristic equation of the block matrix G33 is given by Equation 52.
Where,
If all the roots of the characteristic equation of matrix G33 are negative, then it must satisfy λ1(33)+ λ2(33)<0 and λ1(33)λ2(33)>0, as shown in the derivation process of Equation 53.
From Equation 53, it can be seen that when , all eigenvalues are negative.
If R0 < 1, the no-public opinion equilibrium point E0 is globally asymptotically stable.
Proof. The Lyapunov function is defined as presented in Equation 54.
Its derivative along system Equation 4, as shown in Equation 49
According to Lasalle’s invariance principle, E0 is globally asymptotically stable when R0 < 1.
6 Numerical examples
To demonstrate the model’s validity, numerical simulations were designed based on the system (4). The hyper-degree distribution follows a generalized power-law form: , where b represents the minimum value of ||Ki||. Assuming the range of ||Ki|| in the network spans from 1 to 200, where , . The numerical simulation experiment was set with initial conditions: S (0) = 0.96, I1 (0) = 0.02, I2 (0) = 0.01, and R (0) = 0.01.
6.1 Stability numerical simulation of the no-public opinion equilibrium point
Let α = 0.37, β1 = 0.3, β2 = 0.2, γ = 0.09, ω = 0.375, = 0.065. By Equation 25, we obtain R0 = 0.958 < 1. According to Theorem 5.1, the no-public opinion equilibrium point of the system (7) is locally asymptotically stable.
Figure 4 shows the numerical simulation evolution of population state densities over time t for the system (4) under the condition R0 < 1. The multiple faint lines represent individual simulation trajectories, while the thick solid lines denote the average evolution curves for corresponding variables. HS(t) first declines before rebounding, ultimately stabilizing at a non-zero value. HI1(t) and HI2(t) successively exhibit single peaks, with the peak of HI2(t) l lagging markedly behind that of HI1(t). HR(t) increases monotonically to a saturated state. The system ultimately converges to a stable equilibrium where HI1(t) = HI2(t) = 0 while HS(t) and HR(t) > 0, indicating the establishment of stable herd immunity during transmission. The evolutionary trends of each state variable align with theoretical analysis. The average curves exhibit smoothness with fluctuations within reasonable ranges, validating the model’s stability and convergence under different parameter implementations.
FIGURE 4
Figure 5 shows the density distribution of five population categories within system (4) through a heatmap. Where HS(t) exhibits a pronounced gradient decay, reflecting its continuous depletion during the transmission process. HI1(t) exhibits a concentrated distribution, indicating an earlier infection peak with significant spatial clustering. HI2(t) exhibits relatively dispersed high-density zones, suggesting a more gradual transmission process. HR(t) exhibits that the recovery phase dominates initially before levelling off. The overall infected state HI(t) density comprehensively reflects the spatiotemporal heterogeneity resulting from the superposition of these two infection states.
FIGURE 5
Numerical simulations demonstrate that when the basic reproduction number R0 < 1, the no-public opinion equilibrium point of the system Equation 4 exhibits global attractiveness. Initial transmission proportions were set at (0.01, 0.05, 0.1, 0.2), observing the system’s state evolution trajectory over time.
As evident from the subplots in Figure 6, under parameter conditions where R0 < 1, the no-public opinion equilibrium point exhibits global asymptotic stability. Regardless of the initial state, the phase trajectories of the system demonstrate a clear convergence trend in both two-dimensional and three-dimensional projections, ultimately stabilizing at the no-public opinion equilibrium point. The numerical results are consistent with Theorem 4.2 and Theorem 5.3. That is, when the basic reproduction number R0 is less than 1, regardless of the initial proportion of the population affected by the rumors, the system ultimately converges to the no-public opinion equilibrium point.
FIGURE 6
6.2 Stability numerical simulation of the public opinion equilibrium point
Let α = 0.37, β1 = 0.3, β2 = 0.2, γ = 0.01, ω = 0.001, and = 0.065. By Equation 24, we obtain R0 = 8.618 > 1. According to Theorem 5.2, when parameters ϴ1* and ϴ2* satisfy certain conditions, the public opinion equilibrium point is locally asymptotically stable.
Figure 7 shows the numerical simulation evolution of population state densities over time t for the system (4) under the condition R0 > 1. HS(t) exhibits a typical exponential decay trend. As t increases, the population state density of HS(t) continuously diminishes before stabilizing at a non-zero steady state. HI1(t) and HI2(t) sequentially form single peaks, with the peak of HI2(t) lagging significantly behind that of HI1(t). HR(t) exhibits a monotonically increasing S-shaped growth trend, with a rapid initial growth rate that gradually slows before ultimately reaching a saturated state. The system ultimately converges to a stable equilibrium where HS(t), HI1(t), HI2(t), and HR(t) > 0. This equilibrium state indicates the establishment of a stable herd immunity pattern following the conclusion of transmission. The evolutionary trends of each state variable align with theoretical analysis. The average curves exhibit smoothness with fluctuations within reasonable bounds, validating the model’s stability and convergence under various parameters.
FIGURE 7
Figure 8 shows the density distribution of five population categories within system Equation 4 through a heatmap. HS(t) exhibits a gradient from top-left to bottom-right, indicating that susceptible individuals are progressively depleted over time, leading to a gradual decrease in density. HI1 (t) exhibits a pronounced lateral band of high density at the midpoint of the timeframe, signifying rapid transmission and peak prevalence of HI1 across a substantial proportion of nodes, followed by subsequent attenuation. The high-density regions for HI2(t) emerge slightly later than those for HI1 and display a more dispersed distribution, reflecting the asynchrony and heterogeneity of transmission. HR(t) density exhibits a color gradient from bottom-left to top-right, indicating that the density of recovered individuals accumulates over time, becoming dominant in the later stages. The high-density region of the total infected state HI (t) persists for an extended period, enabling infections to persist within the system and form distinct transmission peaks.
FIGURE 8
Numerical simulations demonstrate that when the basic reproduction number R0 > 1, the public opinion equilibrium point of the system Equation 4 exhibits global attractiveness. Initial transmission proportions were set at (0.01, 0.05, 0.1, 0.2), observing the system’s state evolution trajectory over time.
As shown in the subplots of Figure 9, under parameter conditions where R0 > 1, the evolutionary trajectory of the system ultimately stabilizes at the same public opinion equilibrium point regardless of the initial infection proportion. This indicates that the epidemic will not die out at this stage but instead establishes a persistent endemic state within the population.
FIGURE 9
6.3 Dynamics of public opinion dissemination: Hypergraph versus graph
Under the condition that the basic reproduction number R0 < 1, the dissemination dynamics of system (4) were compared in both hypergraph and ordinary graph network structures.
As shown in Figure 10, under the condition that R0 < 1, regardless of the network structure, the system trajectory ultimately converges towards the no-public-opinion equilibrium point. This numerically confirms the global stability of this equilibrium point within the parameter range. However, transmission within hypergraph structures exhibits characteristics of higher peaks, larger scales, and slower decay. This arises because hyperedges simulate collective interactions, enabling infections to simultaneously affect multiple susceptible nodes within a single time step, thereby amplifying the dissemination effect. By contrast, the pairwise interactions in ordinary graphs constrain both the speed and scope of transmission.
FIGURE 10
Under the condition that the basic reproduction number R0 > 1, the dissemination dynamics of system Equation 4 were compared in both hypergraph and ordinary graph network structures.
As shown in Figure 11, under conditions where R0 > 1, regardless of the network structure, the system trajectory ultimately converges towards the public opinion equilibrium point of public opinion prevalence. Comparative analysis reveals that higher-order interactions within networks significantly accelerate and amplify the dissemination of public opinion. In hypergraph structures, infection peaks are higher, peak attainment occurs more rapidly, and the system’s convergence towards equilibrium is prolonged.
FIGURE 11
To quantify the role of higher-order interactions in opinion spreading, this paper compare the Hyper-S2IR model on two network substrates: a hypergraph, where a hyperedge represents simultaneous group interaction, and a graph, where only pairwise contacts are allowed. Under identical parameter settings and numerical integration protocols, this paper extracts the following outcome metrics from the infection trajectory HI(t): (i) the basic reproduction number R0; (ii) the infection peak HIpeak; (iii) the peak time tpeak; (iv) the final infection level HIfinal = HI(tend); and (v) the maximum propagation velocity Vmax = maxt. Let α = 0.37, γ = 0.09, β1 = 0.3, β2 = 0.2, ω = 0.001. The comparison results are shown in Table 3.
TABLE 3
| Structure | R0 | HIpeak | tpeak | HIfinal | Vmax |
|---|---|---|---|---|---|
| Graph | 156.02 | 0.3414 | 180 | 0.462 | 0.0447 |
| Hypergraph | 5.449 | 0.0962 | 17 | 0.0559 | 0.0005 |
Comparison of spreading outcomes on graph and hypergraph structures.
Under the same parameter setting, the classical graph and the hypergraph exhibit markedly different spreading dynamics. In terms of threshold strength, R0 = 156.0 on the graph but R0 = 5.449 on the hypergraph, indicating that higher-order interactions rescale the effective spreading threshold. At the macroscopic level, the graph case reaches a later and higher peak and ends with a larger final infected level, whereas the hypergraph reaches its peak much earlier with HIpeak = 0.0962, HIfinal = 0.0559, and a substantially different maximum propagation velocity. Overall, the hypergraph structure does not merely shift a single outcome but reshapes the threshold scale, peak timing, and growth-rate characteristics of propagation, leading to a distinct spreading profile compared with the classical graph.
6.4 Analysis of the dissemination impact of state change parameters
This section employs a numerical simulation system to investigate the influence of key parameters within the system
Equation 4on dissemination dynamics.
- 1.
Impact analysis of parameter α
Let
β1= 0.3,
β2= 0.2,
γ= 0.0085,
ω= 0.001, ||
Ki|| = 50. Let
αtake values of 0.20, 0.30, 0.37, 0.45, and 0.55, respectively. The comparative evolution of the densities of
HI1(t),
HI2(t), and
HR(t) is shown in
Figure 12.
- 2.
Impact analysis of parameter γ
FIGURE 12
Let
α= 0.37,
β1= 0.3,
β2= 0.2,
ω= 0.001, ||
Ki|| = 50. Let
γtake values of 0.0015, 0.0030, 0.0045, 0.0060, 0.0075, 0.0085, and 0.01, respectively. The comparative evolution of the densities of
HI1(t),
HI2(t), and
HR(t) is shown in
Figure 13.
- 3.
Impact analysis of parameter ω
FIGURE 13
Let
α= 0.37,
β1= 0.3,
β2= 0.2,
γ= 0.0085, ||
Ki|| = 50. Let
ωtake values of 0.0005, 0.001, 0.005, 0.01, 0.05, 0.1. The comparative evolution of the densities of
HI1(t),
HI2(t), and
HR(t) is shown in
Figure 14.
- 4.
Impact analysis of parameter βn
FIGURE 14
Let
α= 0.37,
ω= 0.001,
γ= 0.0085, ||
Ki|| = 50. Fix
β1and
β2sequentially at 0.2, while varying the other parameter at 0.1, 0.2, 0.3, 0.4, and 0.5. The density evolution of
HI1(t) and
HI2(t) is compared in
Figure 15.
- 5.
Impact analysis of parameter group βn
FIGURE 15
Let
α= 0.37,
ω= 0.001,
γ= 0.0085, ||
Ki|| = 50. Let (
β1,
β2) take values (0.1, 0.1), (0.2, 0.2), (0.3, 0.3), (0.1, 0.2), (0.2, 0.1), (0.1, 0.3), and (0.3, 0.1) respectively. The density evolution comparisons of
HI1(t) and
HI2(t) are illustrated in
Figure 16.
- 6.
Global Sensitivity Analysis
FIGURE 16
A global sensitivity analysis was conducted for {α, γ, β1, β2, ω} using a two-step pipeline. Morris screening was applied first, followed by Sobol variance-based decomposition using the total-effect index ST.
Tables 4, 5 show that γ dominates the variance of key outputs, particularly peak timing, while is consistently the second most influential parameter. contributing notably to -related peak and final outcomes and remaining non-negligible for dynamics. Morris screening provides a consistent ranking, confirming that type-specific peak outputs better reveal the effects of the cross-type conversion and competition parameters.
TABLE 4
| Parameter | HI1peak | t1peak | HI1fianl | V1max | HI2peak | t2peak | HI2fianl | V2max |
|---|---|---|---|---|---|---|---|---|
| α | 0.351 | 0.498 | 0.344 | 0.429 | 0.218 | 0.439 | 0.204 | 0.339 |
| γ | 0.818 | 0.908 | 0.836 | 0.756 | 0.616 | 0.918 | 0.616 | 0.702 |
| β1 | 0.044 | 0.018 | 0.050 | 0.004 | 0.083 | 0.089 | 0.113 | 0.012 |
| β2 | 0.058 | 0.046 | 0.137 | 0.006 | 0.250 | 0.040 | 0.307 | 0.048 |
| ω | 0.022 | 0.038 | 0.021 | 0.011 | 0.022 | 0.026 | 0.023 | 0.011 |
Sobol total effect indices ST for HI1(t) and HI2(t).
TABLE 5
| Output | Metric | α | γ | β1 | β2 | ω |
|---|---|---|---|---|---|---|
| HI1peak | μ | 0.115 | 0.056 | 0.037 | 0.006 | 0.032 |
| σ | 0.200 | 0.120 | 0.092 | 0.016 | 0.095 | |
| HI2peak | μ | 0.084 | 0.031 | 0.030 | 0.006 | 0.016 |
| σ | 0.179 | 0.086 | 0.076 | 0.017 | 0.044 |
Morris screening.
7 Conclusion
7.1 Conclusions and discussion
Based on the construction and analysis of the Hyper-S2IR model rooted in hypergraph theory, this study provides a theoretical framework for explaining opinion diffusion under group interactions. The main contribution is the use of hypergraphs to represent higher-order social contacts, together with two categories of propagators to capture heterogeneity in motivation and influence, thereby modeling phenomena such as the “Goebbels effect” and incentive-driven competition in propagation. The basic reproduction number is derived analytically, yielding a clear survival threshold: when the threshold condition is not met, the system converges to the no-opinion equilibrium, whereas exceeding it leads to persistent diffusion. Comparative experiments between hypergraph and classical graph structures further show that higher-order interactions do not simply change one outcome, but rescale the effective threshold and reshape the full propagation profile, including peak timing, peak level, final reach, and the maximum growth rate, under the same parameter setting. In addition, the global sensitivity analysis identifies the exit-from-spreading and adoption mechanisms as the most influential levers overall, while the competition and conversion mechanisms between the two propagator types become clearly visible when type-specific trajectories are analyzed, especially for the second propagator class.
7.2 Implications for intervention strategies
The global sensitivity analysis clarifies which levers matter most for governance. Across the type-specific outcomes, the recovery or “exit-from-spreading” mechanism is the most influential factor, suggesting that interventions should prioritize accelerating clarification and correction processes, such as rapid fact-checking, timely official responses, and platform actions that reduce continued engagement with misleading content. The infection or adoption mechanism is the next key lever, which supports friction-based measures like “think-before-you-share” prompts, warning labels, and repost delays to reduce impulsive forwarding.
Importantly, using the two spreading types makes the competitive and conversion dynamics visible: the parameters governing competition between the two spreader types meaningfully affect the second type’s peak and final level. This implies that governance should not only suppress misinformation but also strengthen credible counter-narratives, for example, by boosting authoritative accounts and amplifying verified information to counter emotional narratives. Overall, effective strategies combine faster “exit” from spreading, reduced adoption of questionable content, and targeted shaping of the competition between credible and emotional narratives.
7.3 Limitations and future extensions
This paper mainly focuses on the theoretical analysis of the Hyper-S2IR model, including the derivation of the basic reproduction number R0 and the proofs of local and global stability, with numerical simulations used to illustrate the corresponding dynamical behaviors. A first limitation is that spreader heterogeneity is represented by only two propagator types, HI1 and HI2. This stylized dichotomy is adopted for analytical tractability and to isolate the impact of heterogeneity on thresholds and stability, but real public sentiment may involve a broader and even continuous spectrum of stances and behaviors.
A second limitation is that the present study emphasizes mechanism and theory rather than case-specific empirical deployment. While the hypergraph formulation is compatible with constructing hyperedges from group-level interactions and the two propagator types admit behavior-based interpretations, a detailed operational mapping from platform records to hypergraph structure and propagator labels is left for future work. Likewise, systematic parameter calibration and benchmark-style comparisons under real datasets are beyond the scope of the current theoretical contribution.
A third limitation concerns scalability. Our simulations rely on a degree-stratified mean-field implementation that aggregates nodes into hyperdegree classes, and the computational behavior and approximation accuracy for very large-scale networks are not analyzed. Future work will investigate scalable stratification and coarse-graining strategies driven by hyperdegree and hyperedge-size distributions to retain key higher-order effects while improving computational feasibility.
Finally, the framework can be extended to Hyper-SnIR with more propagator classes and can be combined with Multi-Modal Hypergraph Transformer [34] and transformer-based deep learning [35] to support dynamic relation updating and finer interaction modeling, improving applicability to realistic public opinion settings.
Statements
Data availability statement
The raw data supporting the conclusions of this article will be made available by the authors, without undue reservation.
Author contributions
CZ: Conceptualization, Funding acquisition, Resources, Writing – review and editing. XL: Data curation, Formal Analysis, Methodology, Writing – original draft. LL: Investigation, Supervision, Writing – review and editing. JR: Software, Writing – review and editing. JM: Validation, Writing – review and editing. LZ: Visualization, Writing – review and editing.
Funding
The author(s) declared that financial support was received for this work and/or its publication. This research was funded by the Tangshan Science and Technology Project, grant number 24140202C; The Basic Scientific Research Operating Expenses of Provincial Universities, grant number JJC2024036; The Funded by Science Research Project of Hebei Education Department, grant number QN2024252, and the Basic scientific research operating expenses of provincial universities, grant number JJC2024075.
Acknowledgments
The authors would like to thank the editor-in-chief, the editor, and the reviewers for their valuable comments and suggestions.
Conflict of interest
The author(s) declared that this work was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Generative AI statement
The author(s) declared that generative AI was not used in the creation of this manuscript.
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Summary
Keywords
goebbels effect, hypergraph, hyper-S2IR mode, information diffusion, public opinion dissemination
Citation
Zhang C, Li X, Liu L, Ren J, Ma J and Zhang L (2026) Hyper-S2IR: a model for characterizing higher-order interactions and dynamics in public opinion dissemination. Front. Phys. 14:1782845. doi: 10.3389/fphy.2026.1782845
Received
07 January 2026
Revised
07 February 2026
Accepted
24 February 2026
Published
26 March 2026
Volume
14 - 2026
Edited by
Hairong Lin, Central South University, China
Reviewed by
Njitacke Tabekoueng Zeric, University of Buea, Cameroon
Hongsong Chen, University of Science and Technology Beijing, China
Updates
Copyright
© 2026 Zhang, Li, Liu, Ren, Ma and Zhang.
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Lu Liu, liulu_hblg@ncst.edu.cn
Disclaimer
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.